Literature DB >> 15258732

Fluids reverse the early lipopolysaccharide-induced albumin leakage in rodent mesenteric venules.

Peter B Anning1, Simon J Finney, Suveer Singh, C Peter Winlove, Timothy W Evans.   

Abstract

OBJECTIVE: Volume resuscitation is clinically beneficial in patients with sepsis, but few data exist concerning the effects of fluid administration on early events in the inflammatory process. Vascular permeability, leukocyte rolling and leukocyte adhesion in the rodent mesenteric microcirculation were assessed in vivo using intravital microscopy, and the effect of fluid administration on lipopolysaccharide (LPS)-induced changes recorded.
DESIGN: Prospective, repeated measures study.
SETTING: University hospital laboratory.
SUBJECTS: Male Wistar rats in six groups.
INTERVENTIONS: All animals underwent intravital microscopic examination of mesenteric post-capillary venules. LPS or vehicle was applied topically. Animals received either no additional fluids, 0.9% saline (16 ml/kg per h) or 5% human albumin (16 ml/kg per h) commencing 30 min prior to LPS/vehicle administration.
MEASUREMENTS AND MAIN RESULTS: Leukocyte rolling, firm adhesion and blood velocity were observed directly. Vascular permeability was assessed using the flux of fluorescently labelled albumin into the interstitium. LPS significantly increased the median (IQR) number of leukocytes rolling and firmly adherent relative to baseline (at 60 min rolling increased from 12.0 (10.3-13.8) to 40.3 (36.0-47.5) cells/min; adhesion increased from 1 (1-2) to 17 (12-26) cells/100 microm; n=5, p<0.01). Transvascular albumin flux was significantly increased 45 min after LPS application (p<0.01), but not after vehicle. Administration of either 0.9% saline (n=5) or 5% human albumin (n=6), significantly attenuated LPS-induced increases in albumin flux (p<0.05), leukocyte rolling (p<0.01) and adhesion (p<0.01). Fluid administration did not appear to alter shear rates.
CONCLUSIONS: Pre-emptive volume administration with either saline or albumin prevented early LPS-induced microcirculatory changes by an undefined effect that is unrelated to changes in microvascular flow.

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Year:  2004        PMID: 15258732     DOI: 10.1007/s00134-004-2385-3

Source DB:  PubMed          Journal:  Intensive Care Med        ISSN: 0342-4642            Impact factor:   17.440


  27 in total

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4.  Endothelin-1 causes P-selectin-dependent leukocyte rolling and adhesion within rat mesenteric microvessels.

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Review 6.  Physiological and pharmacological evidence for the regulation of permeability.

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7.  Endothelial barrier resistance in multiple organs after septic and nonseptic challenges in the rat.

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8.  Hydroxyethyl starch (130 kD), but not crystalloid volume support, improves microcirculation during normotensive endotoxemia.

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9.  Histamine can induce leukocyte rolling in rat mesenteric venules.

Authors:  K Ley
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10.  Signaling via beta(2) integrins triggers neutrophil-dependent alteration in endothelial barrier function.

Authors:  N Gautam; H Herwald; P Hedqvist; L Lindbom
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2.  Polymicrobial sepsis and endotoxemia promote microvascular thrombosis via distinct mechanisms.

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Review 4.  Microvascular resuscitation as a therapeutic goal in severe sepsis.

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6.  High mixed venous oxygen saturation levels do not exclude fluid responsiveness in critically ill septic patients.

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Review 7.  Effects of volume resuscitation on the microcirculation in animal models of lipopolysaccharide sepsis: a systematic review.

Authors:  Nchafatso G Obonyo; Jonathon P Fanning; Angela S Y Ng; Leticia P Pimenta; Kiran Shekar; David G Platts; Kathryn Maitland; John F Fraser
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  7 in total

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