Literature DB >> 15246642

Amyloid deposition as a cause of atrial remodelling in persistent valvular atrial fibrillation.

O Leone1, G Boriani, B Chiappini, D Pacini, G Cenacchi, S Martin Suarez, C Rapezzi, M L Bacchi Reggiani, G Marinelli.   

Abstract

AIM: The spectrum of histological alterations, namely atrial amyloidosis, in the right and left atria of patients with chronic persistent atrial fibrillation (AF) and rheumatic heart disease is not completely known. METHODS AND
RESULTS: One hundred and twenty-eight atrial appendages (66 left and 62 right), obtained from 72 patients with rheumatic valve disease and chronic AF undergoing cardiac surgery for valve replacement or repair and AF treatment were histologically evaluated for the presence of amyloid deposits. One hundred and four specimens of left and right auricles from 52 patients in sinus rhythm with severe chronic heart failure undergoing heart transplant were also analyzed (controls). Amyloid was found in 33 (46%) valvular patients with chronic persistent AF and in 6 (12%) controls. Amyloid was related to the presence and duration of AF, was more frequently found in left atrial samples and was independent of age. On stepwise logistic regression analysis, AF duration and female gender were independently related to amyloid deposition.
CONCLUSIONS: Patients with long-standing AF and rheumatic heart disease have a very high prevalence of atrial amyloidosis. Amyloid deposition is more frequent in left than in right atrial appendage and correlates with AF duration and female gender. Amyloid deposition could constitute an additional histological feature in the structural remodeling of atria during long-standing AF, at least in rheumatic valve disease. Persistence of AF might play a pivotal role in promoting amyloid deposition.

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Year:  2004        PMID: 15246642     DOI: 10.1016/j.ehj.2004.04.007

Source DB:  PubMed          Journal:  Eur Heart J        ISSN: 0195-668X            Impact factor:   29.983


  23 in total

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9.  Sex Difference in the Risk of Dementia in Patients with Atrial Fibrillation.

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10.  Telocytes in human isolated atrial amyloidosis: ultrastructural remodelling.

Authors:  E Mandache; M Gherghiceanu; C Macarie; S Kostin; L M Popescu
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