Literature DB >> 15189937

Increased arteriovenous carboxyhemoglobin differences in patients with inflammatory pulmonary diseases.

Hiroyasu Yasuda1, Takahiko Sasaki, Mutsuo Yamaya, Satoru Ebihara, Masahiro Maruyama, Akio Kanda, Hidetada Sasaki.   

Abstract

PURPOSE: Exhaled carbon monoxide and arterial blood carboxyhemoglobin concentrations increase in inflammatory pulmonary diseases. The present study was undertaken to elucidate whether arteriovenous carboxyhemoglobin (a-vHb-CO) concentration differences are also useful to define the site of inflammation, either in the lung or organs other than the lung.
MATERIALS AND METHODS: We examined concentrations of carboxyhemoglobin in both arterial and peripheral venous blood and exhaled carbon monoxide in patients with acute pulmonary inflammation including bronchial asthma (n = 18) and pneumonia (n = 33), and those in patients with extrapulmonary inflammatory diseases, including acute pyelonephritis (n = 28) and active rheumatoid arthritis (n = 16).
RESULTS: The values of carboxyhemoglobin in both arterial and peripheral venous blood were significantly higher in patients with pulmonary and extrapulmonary inflammation compared with those in control subjects (n = 22). Furthermore, a-vHb-CO differences in patients with inflammatory pulmonary diseases were higher than those in patients with acute pyelonephritis and patients with rheumatoid arthritis, and than those in control subjects. The a-vHb-CO differences correlated with the WBC count of peripheral venous blood in patients with pneumonia. In patients with bronchial asthma, the a-vHb-CO differences inversely correlated with FEV(1), although they did not correlate with WBC count of peripheral venous blood. The a-vHb-CO differences in patients with acute pyelonephritis were higher than those in patients with active rheumatoid arthritis.
CONCLUSION: The present study suggests that a-vHb-CO differences may be a useful means to define the site of inflammation, either in the lung or organs other than the lung, in patients with a fever of unknown origin. The large a-vHb-CO differences may be caused by carbon monoxide production in pulmonary inflammation.

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Year:  2004        PMID: 15189937     DOI: 10.1378/chest.125.6.2160

Source DB:  PubMed          Journal:  Chest        ISSN: 0012-3692            Impact factor:   9.410


  7 in total

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2.  Association of susceptibility to the development of pneumonia in the older Japanese population with haem oxygenase-1 gene promoter polymorphism.

Authors:  H Yasuda; S Okinaga; M Yamaya; T Ohrui; M Higuchi; M Shinkawa; S Itabashi; K Nakayama; M Asada; A Kikuchi; S Shibahara; H Sasaki
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Review 3.  Carbon monoxide in exhaled breath testing and therapeutics.

Authors:  Stefan W Ryter; Augustine M K Choi
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Review 4.  Asthma across the ages: knowledge gaps in childhood asthma.

Authors:  Stanley J Szefler; James F Chmiel; Anne M Fitzpatrick; George Giacoia; Thomas P Green; Daniel J Jackson; Heber C Nielsen; Wanda Phipatanakul; Hengameh H Raissy
Journal:  J Allergy Clin Immunol       Date:  2013-11-28       Impact factor: 10.793

5.  Normal values of exhaled carbon monoxide in healthy subjects: comparison between two methods of assessment.

Authors:  Umberto Moscato; Andrea Poscia; Riccardo Gargaruti; Giovanni Capelli; Franco Cavaliere
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6.  Exhaled carbon monoxide levels in infants and toddlers with episodic asthma.

Authors:  Yoichiro Ohara; Takahiro Ohara; Koichi Hashimoto; Mitsuaki Hosoya
Journal:  Fukushima J Med Sci       Date:  2020-06-27

7.  Heme Oxygenase Activity Correlates with Serum Indices of Iron Homeostasis in Healthy Nonsmokers.

Authors:  Andrew J Ghio; Dina M Schreinemachers
Journal:  Biomark Insights       Date:  2016-04-19
  7 in total

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