Literature DB >> 15172187

Temporal gene expression following prosthetic arterial grafting.

David J Willis1, Jeffrey A Kalish, Cheng Li, Evan R Deutsch, Mauricio A Contreras, Frank W LoGerfo, William C Quist.   

Abstract

BACKGROUND: Following prosthetic arterial grafting, cytokines and growth factors released within the perianastomotic tissues stimulate smooth muscle cell proliferation and matrix production. While much in vitro work has characterized this response, little understanding exists regarding the sequential up- and down-regulation of genes following prosthetic arterial grafting. This study evaluates temporal gene expression at the distal anastomosis of prosthetic arterial grafts using microarray analysis.
METHODS: Expanded polytetrafluoroethylene (ePTFE) carotid interposition grafts (n = 12) were surgically implanted into mongrel dogs. Distal anastomotic segments were harvested at 7, 14, 30, or 60 days. Contralateral carotid artery served as control. Total RNA was isolated from the anastomotic tissue and paired controls. Samples were probed with oligonucleotide microarrays consisting of approximately 10000 human genes to analyze differential gene expression at each time point.
RESULTS: Forty-nine genes were found to be up-regulated and 37 genes were found to be down-regulated at various time points. Six genes were found to be consistently up-regulated at all time intervals, including collagen type 1 alpha-1 and alpha-2, 80K-L protein (MARCKS), and osteopontin. Six genes were found to be consistently down-regulated, including smoothelin and tropomyosin 2. RT-PCR and immunohistochemistry confirmed the microarray data.
CONCLUSIONS: This study uses microarray analysis to identify genes that were temporally up- and down-regulated after prosthetic arterial grafting. Genes with similar patterns of expression have been identified, providing insights into related cellular pathways that may result in the formation of anastomotic intimal hyperplasia.

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Year:  2004        PMID: 15172187     DOI: 10.1016/j.jss.2003.12.014

Source DB:  PubMed          Journal:  J Surg Res        ISSN: 0022-4804            Impact factor:   2.192


  9 in total

1.  The myristoylated alanine-rich C kinase substrate differentially regulates kinase interacting with stathmin in vascular smooth muscle and endothelial cells and potentiates intimal hyperplasia formation.

Authors:  Dan Yu; Ramkishore Gernapudi; Charles Drucker; Rajabrata Sarkar; Areck Ucuzian; Thomas S Monahan
Journal:  J Vasc Surg       Date:  2019-03-28       Impact factor: 4.268

2.  Gene silencing in human aortic smooth muscle cells induced by PEI-siRNA complexes released from dip-coated electrospun poly(ethylene terephthalate) grafts.

Authors:  Christoph S Nabzdyk; Maggie C Chun; Hunter S Oliver-Allen; Saif G Pathan; Matthew D Phaneuf; Jin-Oh You; Leena K Pradhan-Nabzdyk; Frank W LoGerfo
Journal:  Biomaterials       Date:  2014-01-04       Impact factor: 12.479

3.  Scaffold-free in vitro arterial mimetics: the importance of smooth muscle-endothelium contact.

Authors:  Somali Chaterji; Kinam Park; Alyssa Panitch
Journal:  Tissue Eng Part A       Date:  2010-06       Impact factor: 3.845

4.  MARCKS silencing differentially affects human vascular smooth muscle and endothelial cell phenotypes to inhibit neointimal hyperplasia in saphenous vein.

Authors:  Thomas S Monahan; Nicholas D Andersen; Michelle C Martin; Junaid Y Malek; Gautam V Shrikhande; Leena Pradhan; Christiane Ferran; Frank W LoGerfo
Journal:  FASEB J       Date:  2008-10-21       Impact factor: 5.191

5.  Heparin/Collagen-REDV Modification of Expanded Polytetrafluoroethylene Improves Regional Anti-thrombosis and Reduces Foreign Body Reactions in Local Tissues.

Authors:  Yaping Shan; Gang Chen; Qiqi Shi; Jiaxi Huang; Yaping Mi; Wenbo Zhang; Huifeng Zhang; Bing Jia
Journal:  Front Bioeng Biotechnol       Date:  2022-08-04

Review 6.  ECM-based materials in cardiovascular applications: Inherent healing potential and augmentation of native regenerative processes.

Authors:  Anna V Piterina; Aidan J Cloonan; Claire L Meaney; Laura M Davis; Anthony Callanan; Michael T Walsh; Tim M McGloughlin
Journal:  Int J Mol Sci       Date:  2009-11-20       Impact factor: 6.208

7.  MARCKS Signaling Differentially Regulates Vascular Smooth Muscle and Endothelial Cell Proliferation through a KIS-, p27kip1- Dependent Mechanism.

Authors:  Dan Yu; George Makkar; Tuo Dong; Dudley K Strickland; Rajabrata Sarkar; Thomas Stacey Monahan
Journal:  PLoS One       Date:  2015-11-03       Impact factor: 3.240

8.  Myristoylated Alanine-Rich Protein Kinase Substrate (MARCKS) Regulates Small GTPase Rac1 and Cdc42 Activity and Is a Critical Mediator of Vascular Smooth Muscle Cell Migration in Intimal Hyperplasia Formation.

Authors:  Dan Yu; George Makkar; Dudley K Strickland; Thomas A Blanpied; Deborah J Stumpo; Perry J Blackshear; Rajabrata Sarkar; Thomas S Monahan
Journal:  J Am Heart Assoc       Date:  2015-10-08       Impact factor: 5.501

9.  Delivery of targeted gene therapies using a hybrid cryogel-coated prosthetic vascular graft.

Authors:  Frank LoGerfo; Leena Pradhan-Nabzdyk; Cindy Huynh; Ting-Yu Shih; Alexander Mammoo; Amruta Samant; Saif Pathan; David W Nelson; Christiane Ferran; David Mooney
Journal:  PeerJ       Date:  2019-08-20       Impact factor: 2.984

  9 in total

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