Literature DB >> 15143480

Differential effects of varying concentrations of clostridium difficile toxin A on epithelial barrier function and expression of cytokines.

Shawinder S Johal1, Katie Solomon, Sue Dodson, S Peter Borriello, Yashwant R Mahida.   

Abstract

BACKGROUND: Presentation after Clostridium difficile infection may depend on the level of epithelial exposure to toxins. We investigated epithelial barrier function and expression of interleukin (IL)-8 and transforming growth factor (TGF)-beta in response to varying concentrations of C. difficile toxin A.
METHODS: T84 cells were either preexposed or continuously exposed to C. difficile toxin A (0.01-1000 ng/mL). Barrier function was assessed by measurements of transepithelial electrical resistance.
RESULTS: Preexposure to < or =10 ng/mL toxin A led to an increase in the release of TGF-beta 1, but there was no change in the expression of IL-8. In contrast, after preexposure to >10 ng/mL toxin A, there was enhanced expression of IL-8, but release of TGF-beta 1 was similar to that in control monolayers. After preexposure to >10 ng/mL toxin A, there was complete and irreversible loss of electrical resistance. At lower concentrations, loss of resistance across monolayers was followed by recovery, which was enhanced by all 3 recombinant isoforms of TGF-beta. Pretreatment with recombinant isoforms of TGF-beta or coculture with TGF-beta 3-expressing colonic subepithelial myofibroblasts was also protective.
CONCLUSIONS: In C. difficile infection, the development and severity of colonic inflammation may depend on the exposure of intestinal epithelial cells to toxins and the expression of proinflammatory (IL-8) and protective (TGF-beta) factors.

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Year:  2004        PMID: 15143480     DOI: 10.1086/386287

Source DB:  PubMed          Journal:  J Infect Dis        ISSN: 0022-1899            Impact factor:   5.226


  24 in total

1.  Clostridium difficile toxins induce VEGF-A and vascular permeability to promote disease pathogenesis.

Authors:  Jun Huang; Ciarán P Kelly; Kyriaki Bakirtzi; Javier A Villafuerte Gálvez; Dena Lyras; Steven J Mileto; Sarah Larcombe; Hua Xu; Xiaotong Yang; Kelsey S Shields; Weishu Zhu; Yi Zhang; Jeffrey D Goldsmith; Ishan J Patel; Joshua Hansen; Meijin Huang; Seppo Yla-Herttuala; Alan C Moss; Daniel Paredes-Sabja; Charalabos Pothoulakis; Yatrik M Shah; Jianping Wang; Xinhua Chen
Journal:  Nat Microbiol       Date:  2018-12-03       Impact factor: 17.745

2.  Hypoxia-inducible factor signaling provides protection in Clostridium difficile-induced intestinal injury.

Authors:  Simon A Hirota; Kyla Fines; Jeffrey Ng; Danya Traboulsi; Josh Lee; Eikichi Ihara; Yan Li; William G Willmore; Daniel Chung; Melanie M Scully; Thomas Louie; Shaun Medlicott; Manigandan Lejeune; Kris Chadee; Glen Armstrong; Sean P Colgan; Daniel A Muruve; Justin A MacDonald; Paul L Beck
Journal:  Gastroenterology       Date:  2010-03-27       Impact factor: 22.682

3.  Murine model of Clostridium difficile infection with aged gnotobiotic C57BL/6 mice and a BI/NAP1 strain.

Authors:  S W Pawlowski; G Calabrese; G L Kolling; J Platts-Mills; R Freire; C AlcantaraWarren; B Liu; R B Sartor; R L Guerrant
Journal:  J Infect Dis       Date:  2010-10-26       Impact factor: 5.226

4.  Clostridium difficile toxins A and B decrease intestinal SLC26A3 protein expression.

Authors:  Hayley Coffing; Shubha Priyamvada; Arivarasu N Anbazhagan; Christine Salibay; Melinda Engevik; James Versalovic; Mary Beth Yacyshyn; Bruce Yacyshyn; Sangeeta Tyagi; Seema Saksena; Ravinder K Gill; Waddah A Alrefai; Pradeep K Dudeja
Journal:  Am J Physiol Gastrointest Liver Physiol       Date:  2018-03-29       Impact factor: 4.052

5.  Transforming Growth Factor β1/SMAD Signaling Pathway Activation Protects the Intestinal Epithelium from Clostridium difficile Toxin A-Induced Damage.

Authors:  Christianne Maria Tinoco-Veras; Ana Angélica Q A Santos; Joice Stipursky; Marcelo Meloni; Ana Paula Bérgamo Araujo; Danielle Abreu Foschetti; Diana López-Ureña; Carlos Quesada-Gómez; Renata F C Leitão; Flávia Carvalho Alcantara Gomes; Gerly Anne de Castro Brito
Journal:  Infect Immun       Date:  2017-09-20       Impact factor: 3.441

6.  Identification of a novel virulence factor in Clostridium difficile that modulates toxin sensitivity of cultured epithelial cells.

Authors:  Masashi Miura; Haru Kato; Osamu Matsushita
Journal:  Infect Immun       Date:  2011-07-11       Impact factor: 3.441

7.  Monocytes are highly sensitive to clostridium difficile toxin A-induced apoptotic and nonapoptotic cell death.

Authors:  K Solomon; J Webb; N Ali; R A Robins; Y R Mahida
Journal:  Infect Immun       Date:  2005-03       Impact factor: 3.441

8.  Role of retinol in protecting epithelial cell damage induced by Clostridium difficile toxin A.

Authors:  Andressa A F L Maciel; Reinaldo B Oriá; Manuel B Braga-Neto; Andréa B Braga; Eunice B Carvalho; Herene B M Lucena; Gerly A C Brito; Richard L Guerrant; Aldo A M Lima
Journal:  Toxicon       Date:  2007-07-31       Impact factor: 3.033

9.  Clostridium difficile toxin B-induced colonic inflammation is mediated by the FOXO3/PPM1B pathway in fetal human colon epithelial cells.

Authors:  Qingqing Xu; Ying Li; Yuejuan Zheng; Yijian Chen; Xiaogang Xu; Minggui Wang
Journal:  Am J Transl Res       Date:  2020-10-15       Impact factor: 4.060

10.  Essential role of toxin A in C. difficile 027 and reference strain supernatant-mediated disruption of Caco-2 intestinal epithelial barrier function.

Authors:  P A Sutton; S Li; J Webb; K Solomon; J Brazier; Y R Mahida
Journal:  Clin Exp Immunol       Date:  2008-05-26       Impact factor: 4.330

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