Literature DB >> 15138896

Paroxetine in major depression: correlating plasma concentrations and clinical response.

Claus Normann1, Marion Hörn, Barbara Hummel, Heinz Grunze, Jörg Walden.   

Abstract

BACKGROUND: In analogy to tricyclic antidepressants, serum concentrations of selective serotonin reuptake inhibitors (SSRIs) are frequently measured in order to optimize treatment results. However, clinical evidence for this approach is sparse.
METHODS: Forty patients with major depression were treated with paroxetine 20 mg/day for 14 days and with 40 mg/day for further 49 days. Treatment response measured by Hamilton depression rating scales (HAMD) was correlated with paroxetine plasma concentrations.
RESULTS: There was a significant difference between paroxetine plasma levels at 20 and 40 mg/day, respectively [20 mg/d: median 24 (range 4-358); 40 mg/d: 92 (30-398)]. However, the interindividual variance was very large. 18 out of 40 patients responded to paroxetine treatment.
CONCLUSIONS: Receiver operated characteristic (ROC) analysis suggested no upper or lower limit of response. Responder had significantly higher paroxetine levels at day 7 [responder: 33 (4-107); non-responder: 13 (3-77)] but not at the end of the study [responder 93 (30-361); non-responder: 94 (59-398)]. Furthermore, plasma levels were not related to adverse events, age, body weight or severity of depression. These findings do not support any need for a routine screening of paroxetine plasma concentrations in clinical practice.

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Year:  2004        PMID: 15138896     DOI: 10.1055/s-2004-818990

Source DB:  PubMed          Journal:  Pharmacopsychiatry        ISSN: 0176-3679            Impact factor:   5.788


  7 in total

1.  Serotonin transporter occupancy induced by paroxetine in patients with major depression disorder: a 123I-ADAM SPECT study.

Authors:  Ana M Catafau; Victor Perez; Pedro Plaza; Juan-Carlos Pascual; Santiago Bullich; Marina Suarez; Maria M Penengo; Iluminada Corripio; Dolors Puigdemont; Monica Danus; Javier Perich; Enric Alvarez
Journal:  Psychopharmacology (Berl)       Date:  2006-10-11       Impact factor: 4.530

2.  P-glycoprotein inhibition increases the brain distribution and antidepressant-like activity of escitalopram in rodents.

Authors:  Fionn E O'Brien; Richard M O'Connor; Gerard Clarke; Timothy G Dinan; Brendan T Griffin; John F Cryan
Journal:  Neuropsychopharmacology       Date:  2013-05-14       Impact factor: 7.853

3.  Serotonin transporter genotype interacts with paroxetine plasma levels to influence depression treatment response in geriatric patients.

Authors:  Francis E Lotrich; Bruce G Pollock; Margaret Kirshner; Robert F Ferrell; Charles F Reynolds Iii
Journal:  J Psychiatry Neurosci       Date:  2008-03       Impact factor: 6.186

4.  Exposure-outcome analysis in depressed patients treated with paroxetine using population pharmacokinetics.

Authors:  Jung-Ryul Kim; Hye In Woo; Mi-Ryung Chun; Shinn-Won Lim; Hae Deun Kim; Han Sung Na; Myeon Woo Chung; Woojae Myung; Soo-Youn Lee; Doh Kwan Kim
Journal:  Drug Des Devel Ther       Date:  2015-09-16       Impact factor: 4.162

5.  Possible impact of the CYP2D6*10 polymorphism on the nonlinear pharmacokinetic parameter estimates of paroxetine in Japanese patients with major depressive disorders.

Authors:  Junji Saruwatari; Hiroo Nakashima; Shoko Tsuchimine; Miki Nishimura; Naoki Ogusu; Norio Yasui-Furukori
Journal:  Pharmgenomics Pers Med       Date:  2014-05-28

6.  The influence of 5-HTTLPR genotype on the association between the plasma concentration and therapeutic effect of paroxetine in patients with major depressive disorder.

Authors:  Tetsu Tomita; Norio Yasui-Furukori; Taku Nakagami; Shoko Tsuchimine; Masamichi Ishioka; Ayako Kaneda; Norio Sugawara; Sunao Kaneko
Journal:  PLoS One       Date:  2014-05-23       Impact factor: 3.240

Review 7.  Pharmacogenetics of antidepressant response: an update.

Authors:  Antonio Drago; Diana De Ronchi; Alessandro Serretti
Journal:  Hum Genomics       Date:  2009-04       Impact factor: 4.639

  7 in total

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