Literature DB >> 15133867

mRNA expression profiling reveals a role of Helicobacter pylori vacuolating toxin in escaping host defense.

Jian-Ping Yuan1, Tao Li, Zhen-Hong Li, Gui-Zhen Yang, Bao-Yu Hu, Xiao-Dong Shi, Tie-Liu Shi, Shan-Qing Tong, Xiao-Kui Guo.   

Abstract

AIM: To study the immune response of host to Helicobacter pylori VacA.
METHODS: The monocyte/macrophage-like U937 cells were infected with Helicobacter pylori vacA-positive strain NCTC 11638 or isogenic vacA-negative mutant. Differentially expressed genes were identified at 2, 6, 10, and 24 h post-infection by cDNA microarray. Differential expressions of some genes were confirmed by Northern blot.
RESULTS: More than 100 genes altered their mRNA expression at different time points respectively, many of which were identified to be related to immune evasion.
CONCLUSION: VacA is a crucial element for H pylori to escape from host immune defense by means of differentially regulating the expression of some related genes. These genes, previously known or unknown to be involved in the mechanism of immune evasion, deserve further investigation to unearth much more information complicated in the immune response.

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Year:  2004        PMID: 15133867      PMCID: PMC4656298          DOI: 10.3748/wjg.v10.i10.1528

Source DB:  PubMed          Journal:  World J Gastroenterol        ISSN: 1007-9327            Impact factor:   5.742


  45 in total

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Review 7.  Interactions between major histocompatibility complex class II surface expression and HIV: implications for pathogenesis.

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Review 10.  The immune response against Helicobacter pylori--a direct linkage to the development of gastroduodenal disease.

Authors:  A Ibraghimov; J Pappo
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  4 in total

Review 1.  Chemical compounds from anthropogenic environment and immune evasion mechanisms: potential interactions.

Authors:  Julia Kravchenko; Emanuela Corsini; Marc A Williams; William Decker; Masoud H Manjili; Takemi Otsuki; Neetu Singh; Faha Al-Mulla; Rabeah Al-Temaimi; Amedeo Amedei; Anna Maria Colacci; Monica Vaccari; Chiara Mondello; A Ivana Scovassi; Jayadev Raju; Roslida A Hamid; Lorenzo Memeo; Stefano Forte; Rabindra Roy; Jordan Woodrick; Hosni K Salem; Elizabeth P Ryan; Dustin G Brown; William H Bisson; Leroy Lowe; H Kim Lyerly
Journal:  Carcinogenesis       Date:  2015-05-22       Impact factor: 4.944

2.  NF-kappaB and ERK-signaling pathways contribute to the gene expression induced by cag PAI-positive-Helicobacter pylori infection.

Authors:  Wataru Shibata; Yoshihiro Hirata; Haruhiko Yoshida; Motoyuki Otsuka; Yujin Hoshida; Keiji Ogura; Shin Maeda; Tomoya Ohmae; Ayako Yanai; Yuzo Mitsuno; Naohiko Seki; Takao Kawabe; Masao Omata
Journal:  World J Gastroenterol       Date:  2005-10-21       Impact factor: 5.742

3.  Gene expression profiling in gastric mucosa from Helicobacter pylori-infected and uninfected patients undergoing chronic superficial gastritis.

Authors:  Ze-Min Yang; Wei-Wen Chen; Ying-Fang Wang
Journal:  PLoS One       Date:  2012-03-16       Impact factor: 3.240

4.  Helicobacter pylori sabA gene is associated with iron deficiency anemia in childhood and adolescence.

Authors:  Seiichi Kato; Takako Osaki; Shigeru Kamiya; Xue-Song Zhang; Martin J Blaser
Journal:  PLoS One       Date:  2017-08-30       Impact factor: 3.240

  4 in total

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