Literature DB >> 15088994

Adventitial response to intravascular brachytherapy in a rabbit model of restenosis.

Paul Wexberg1, Konrad Mück, Ursula Windberger, Susanna Lang, Martin Osranek, Franz Weidinger, Gerald Maurer, Michael Gottsauner-Wolf.   

Abstract

BACKGROUND: The incidence of late major adverse cardiac events (MACE) after coronary brachytherapy is higher than in controls. Because expansive remodeling has been shown to correlate with poor clinical outcome after vascular interventions, we studied adventitial changes after intravascular irradiation in a rabbit model.
METHODS: Twenty normolipidemic rabbits underwent balloon injury in both external iliac arteries. One artery was assigned for subsequent irradiation with a 90Y source (15 Gy or 30 Gy at 0.5 mm in the vessel wall). After four weeks morphometric measurements were made and cell density and collagen amount determined. Staining for Ki67 identified proliferating cells; apoptotic cells were identified by TUNEL staining. Proliferative and apoptotic indices were calculated as the number of respective positive cells/total cell count x100.
RESULTS: The neointimal area decreased to 0.27 +/- 0.3 mm2 after irradiation compared with 0.55 +/- 0.2 mm2 in controls (p=0.007), whereas adventitial area increased from 0.62 +/- 0.3 mm2 to 0.87 +/- 0.3 mm2 (p=0.02). Irradiation reduced both the proliferative (0.95 +/- 2.6 vs. 3.73 +/- 4.7, p=0.026) and apoptotic (0.006 +/- 0.02 vs. 0.107 +/- 0.2, p=0.03) indices in the neointima, but not in the other arterial-wall layers. Collagen amount and arterial remodeling did not differ between the groups. There was no difference between 15 and 30 Gy in any of the parameters, although adventitial thickening was more pronounced in the high-dose group.
CONCLUSIONS: In normolipidemic rabbits, intravascular beta-irradiation after balloon angioplasty is associated with an increase in neoadventitia and a reduction of neointima. It is conceivable that this phenomenon may contribute to the increased incidence of late MACE after vascular brachytherapy.

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Year:  2004        PMID: 15088994     DOI: 10.1007/bf03040486

Source DB:  PubMed          Journal:  Wien Klin Wochenschr        ISSN: 0043-5325            Impact factor:   1.704


  30 in total

1.  Intraarterial beta irradiation induces smooth muscle cell apoptosis and reduces medial cellularity in a hypercholesterolemic rabbit restenosis model.

Authors:  V Verin; Y Popowski; M L Bochaton-Piallat; J Belenger; P Urban; P Neuville; M Redard; M Costa; G Celetta; G Gabbiani
Journal:  Int J Radiat Oncol Biol Phys       Date:  2000-02-01       Impact factor: 7.038

2.  Prescribing, recording, and reporting in endovascular brachytherapy. Quality assurance, equipment, personnel and education.

Authors:  R Pötter; E Van Limbergen; W Dries; Y Popowski; V Coen; C Fellner; D Georg; C Kirisits; P Levendag; H Marijnissen; H Marsiglia; J J Mazeron; B Pokrajac; P Scalliet; V Tamburini
Journal:  Radiother Oncol       Date:  2001-06       Impact factor: 6.280

3.  Prolonged antiplatelet therapy to prevent late thrombosis after intracoronary gamma-radiation in patients with in-stent restenosis: Washington Radiation for In-Stent Restenosis Trial plus 6 months of clopidogrel (WRIST PLUS).

Authors:  R Waksman; A E Ajani; R L White; E Pinnow; R Dieble; A B Bui; M Taaffe; L Gruberg; G S Mintz; L F Satler; A D Pichard; K K Kent; J Lindsay
Journal:  Circulation       Date:  2001-05-15       Impact factor: 29.690

4.  Effect of intravascular irradiation on cell proliferation, apoptosis, and vascular remodeling after balloon overstretch injury of porcine coronary arteries.

Authors:  R Waksman; J C Rodriguez; K A Robinson; G D Cipolla; I R Crocker; N A Scott; S B King; J N Wilcox
Journal:  Circulation       Date:  1997-09-16       Impact factor: 29.690

5.  The radiobiology of intravascular irradiation.

Authors:  D J Brenner; R C Miller; E J Hall
Journal:  Int J Radiat Oncol Biol Phys       Date:  1996-11-01       Impact factor: 7.038

6.  Arterial remodeling after experimental percutaneous injury is highly dependent on adventitial injury and histopathology.

Authors:  M E Staab; S S Srivatsa; A Lerman; G Sangiorgi; M H Jeong; W D Edwards; D R Holmes; R S Schwartz
Journal:  Int J Cardiol       Date:  1997-01-03       Impact factor: 4.164

7.  The effect of endovascular irradiation on platelet recruitment at sites of balloon angioplasty in pig coronary arteries.

Authors:  M Y Salame; S Verheye; S P Mulkey; N A Chronos; S B King; I R Crocker; K A Robinson
Journal:  Circulation       Date:  2000-03-14       Impact factor: 29.690

8.  Hematopoietic stem cells differentiate into vascular cells that participate in the pathogenesis of atherosclerosis.

Authors:  Masataka Sata; Akio Saiura; Atsushi Kunisato; Akihiro Tojo; Seiji Okada; Takeshi Tokuhisa; Hisamaru Hirai; Masatoshi Makuuchi; Yasunobu Hirata; Ryozo Nagai
Journal:  Nat Med       Date:  2002-04       Impact factor: 53.440

9.  Adventitial myofibroblasts contribute to neointimal formation in injured porcine coronary arteries.

Authors:  Y Shi; J E O'Brien; A Fard; J D Mannion; D Wang; A Zalewski
Journal:  Circulation       Date:  1996-10-01       Impact factor: 29.690

Review 10.  Regulation of smooth muscle cell proliferation and its possible role in preventing restenosis post-angioplasty.

Authors:  S Nikol; B Höfling
Journal:  Wien Klin Wochenschr       Date:  1995       Impact factor: 1.704

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  3 in total

1.  Opposite effects of vascular irradiation on inflammatory response and apoptosis induction in the vessel wall layers via the peroxynitrite-poly(ADP-ribose) polymerase pathway.

Authors:  Carsten J Beller; Eszter Horvath; Jens Kosse; Alexander Becker; Tamás Radovits; Robert Krempien; Irina Berger; Siegfried Hagl; Csaba Szabó; Gábor Szabó
Journal:  Clin Res Cardiol       Date:  2006-10-10       Impact factor: 5.460

2.  Phenotypic transformation and migration of adventitial cells following angioplasty.

Authors:  Yong-Li Wang; Li-Zhen Liu; Zhong-Hui He; Kun-Hong Ding; Feng Xue
Journal:  Exp Ther Med       Date:  2012-04-17       Impact factor: 2.447

Review 3.  Radiation-induced cardiac side-effects: The lung as target for interacting damage and intervention.

Authors:  Julia Wiedemann; Robert P Coppes; Peter van Luijk
Journal:  Front Oncol       Date:  2022-07-22       Impact factor: 5.738

  3 in total

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