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Literature DB >> 15047927

The molecular perspective: cytochrome C and apoptosis.

David S Goodsell¹.

Abstract

Entities: Gene

Mesh：

Substances：
Cytochromes c

Year: 2004 PMID： 15047927 DOI： 10.1634/theoncologist.9-2-226

Source DB: PubMed Journal: Oncologist ISSN： 1083-7159

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8 in total

1. Resveratrol protects against arsenic trioxide-induced oxidative damage through maintenance of glutathione homeostasis and inhibition of apoptotic progression.

Authors: Chengzhi Chen; Xuejun Jiang; Yanhao Lai; Yuan Liu; Zunzhen Zhang
Journal: Environ Mol Mutagen Date: 2014-10-23 Impact factor: 3.216

Review 2. Programmed cell death in plants: A chloroplastic connection.

Authors: Vivek Ambastha; Baishnab C Tripathy; Budhi Sagar Tiwari
Journal: Plant Signal Behav Date: 2015

3. Penetration of lysozyme and cytochrome C in lipid bilayer: fluorescent study.

Authors: Ivaylo Zlatanov; Antoaneta Popova
Journal: J Membr Biol Date: 2011-07-08 Impact factor: 1.843

4. Förster Resonance Energy Transfer Study of Cytochrome c-Lipid Interactions.

Authors: Galyna P Gorbenko; Valeriya Trusova; Julian G Molotkovsky
Journal: J Fluoresc Date: 2017-09-06 Impact factor: 2.217

5. Glucocorticoid-induced tumor necrosis factor receptor negatively regulates activation of human primary natural killer (NK) cells by blocking proliferative signals and increasing NK cell apoptosis.

Authors: Baoying Liu; Zhuqing Li; Sankaranarayana P Mahesh; Seth Pantanelli; Frank S Hwang; Willie O Siu; Robert B Nussenblatt
Journal: J Biol Chem Date: 2008-01-29 Impact factor: 5.157

The molecular perspective: cytochrome C and apoptosis.

1. Resveratrol protects against arsenic trioxide-induced oxidative damage through maintenance of glutathione homeostasis and inhibition of apoptotic progression.

Review 2. Programmed cell death in plants: A chloroplastic connection.

3. Penetration of lysozyme and cytochrome C in lipid bilayer: fluorescent study.

4. Förster Resonance Energy Transfer Study of Cytochrome c-Lipid Interactions.

5. Glucocorticoid-induced tumor necrosis factor receptor negatively regulates activation of human primary natural killer (NK) cells by blocking proliferative signals and increasing NK cell apoptosis.

6. Anti-apoptotic actions of vasopressin in H32 neurons involve MAP kinase transactivation and Bad phosphorylation.

7. Mitochondrial cytochrome c release: a factor to consider in mitochondrial disease?

Review 8. The cancer cell's "power plants" as promising therapeutic targets: an overview.