Literature DB >> 15023809

Increased risk for Alzheimer disease with the interaction of MPO and A2M polymorphisms.

Mario Zappia1, Ida Manna, Paolo Serra, Rita Cittadella, Virginia Andreoli, Antonella La Russa, Fernanda Annesi, Patrizia Spadafora, Nelide Romeo, Giuseppe Nicoletti, Demetrio Messina, Antonio Gambardella, Aldo Quattrone.   

Abstract

BACKGROUND: The genes encoding myeloperoxidase (MPO) and alpha(2)-macroglobulin (A2M) are involved in molecular pathways leading to beta-amyloid deposition. Two polymorphic sites in these genes (MPO-G/A and A2M-Ile/Val) have been associated with Alzheimer disease (AD), but conflicting findings have been reported in populations with different ethnic backgrounds.
OBJECTIVES: To study the association of MPO-G/A and A2M-Ile/Val polymorphisms with sporadic AD and to investigate the interactions among the MPO, A2M, and apolipoprotein E (APOE) gene polymorphisms in determining the risk of the development of AD.
DESIGN: Case-control study.
SETTING: Referral center for AD in Calabria, southern Italy. PARTICIPANTS: One hundred forty-eight patients with sporadic AD and 158 healthy control subjects.
RESULTS: The MPO-G and A2M-Val alleles were found more frequently in cases than in controls, as were the MPO-G/G and A2M-Val/Val genotypes. The odds ratio (OR) for the MPO-G/G genotype was 1.78 (95% confidence interval [CI], 1.13-2.80); for the A2M-Val/Val genotype, 3.81 (95% CI, 1.66-8.75). The presence of MPO-G/G and A2M-Val/Val genotypes synergistically increased the risk of AD (OR, 25.5; 95% CI, 4.65-139.75). Stratification of cases by sex, age at onset of AD, and APOE-epsilon 4 status did not show significant differences in the distribution of MPO or A2M polymorphisms.
CONCLUSIONS: The MPO and A2M polymorphisms are associated with sporadic AD in southern Italy. Moreover, a genomic interaction between these polymorphisms increases the risk of the development of AD.

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Year:  2004        PMID: 15023809     DOI: 10.1001/archneur.61.3.341

Source DB:  PubMed          Journal:  Arch Neurol        ISSN: 0003-9942


  15 in total

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