Literature DB >> 14977574

Sepsis associated encephalopathy (SAE): a review.

Rebecca Green1, L Keith Scott, Alireza Minagar, Steven Conrad.   

Abstract

Sepsis associated encephalopathy (SAE) is a poorly understood condition that is associated with severe sepsis and appears to have a negative influence on survival. The incidence of encephalopathy secondary to sepsis is unknown. Amino acid derangements, blood-brain barrier disruption, abnormal neurotransmitters, and direct CNS effect are possible causes of septic encephalopathy. Research has not defined the pathogenesis of SAE.

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Year:  2004        PMID： 14977574     DOI： 10.2741/1250

Source DB:  PubMed          Journal:  Front Biosci        ISSN： 1093-4715


  18 in total

1.  Effects of hydrogen sulfide on a rat model of sepsis-associated encephalopathy.

Authors:  Di Chen; Hao Pan; Chunwen Li; Xiucai Lan; Beibei Liu; Guangtian Yang
Journal:  J Huazhong Univ Sci Technolog Med Sci       Date:  2011-10-25

2.  Cronobacter sakazakii Infection in Early Postnatal Rats Impaired Contextual-Associated Learning: a Putative Role of C5a-Mediated NF-κβ and ASK1 Pathways.

Authors:  Ponnusamy Vinay; Christopher Karen; Krishnaswamy Balamurugan; Koilmani Emmanuvel Rajan
Journal:  J Mol Neurosci       Date:  2020-06-21       Impact factor: 3.444

3.  [Early alterations in rat brain protein expression during sepsis].

Authors:  J Hinkelbein; A Kalenka; R E Feldmann
Journal:  Anaesthesist       Date:  2009-02       Impact factor: 1.041

4.  TNF is a key mediator of septic encephalopathy acting through its receptor, TNF receptor-1.

Authors:  Jessy J Alexander; Alexander Jacob; Patrick Cunningham; Lauren Hensley; Richard J Quigg
Journal:  Neurochem Int       Date:  2007-08-17       Impact factor: 3.921

5.  TRIL is involved in cytokine production in the brain following Escherichia coli infection.

Authors:  Paulina Wochal; Vijay A K Rathinam; Aisling Dunne; Thaddeus Carlson; Wen Kuang; Katherine J Seidl; J Perry Hall; Lih-Ling Lin; Mary Collins; Stefan A Schattgen; Christopher R MacKay; Caio T Fagundes; Susan Carpenter; Katherine A Fitzgerald; Luke A J O'Neill
Journal:  J Immunol       Date:  2014-07-11       Impact factor: 5.422

6.  Endotoxin-induced gene expression differences in the brain and effects of iNOS inhibition and norepinephrine.

Authors:  Stephanie Wolff; Sabine Klatt; Jens C Wolff; Jochen Wilhelm; Ludger Fink; Manfred Kaps; Bernhard Rosengarten
Journal:  Intensive Care Med       Date:  2009-01-21       Impact factor: 17.440

Review 7.  What we know: the inflammatory basis of hepatic encephalopathy.

Authors:  K Milewski; M Oria
Journal:  Metab Brain Dis       Date:  2015-10-26       Impact factor: 3.584

8.  Lipopolysaccharide-induced interleukin-6 production is controlled by glycogen synthase kinase-3 and STAT3 in the brain.

Authors:  Eléonore Beurel; Richard S Jope
Journal:  J Neuroinflammation       Date:  2009-03-11       Impact factor: 8.322

9.  Effects of inducible nitric oxide synthase inhibition or norepinephrine on the neurovascular coupling in an endotoxic rat shock model.

Authors:  Bernhard Rosengarten; Stephanie Wolff; Sabine Klatt; Ralf T Schermuly
Journal:  Crit Care       Date:  2009-08-26       Impact factor: 9.097

10.  Effects of anti-inflammatory vagus nerve stimulation on the cerebral microcirculation in endotoxinemic rats.

Authors:  Stanka Mihaylova; Anke Killian; Konstantin Mayer; Soni Savai Pullamsetti; Ralph Schermuly; Bernhard Rosengarten
Journal:  J Neuroinflammation       Date:  2012-07-25       Impact factor: 8.322
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