Literature DB >> 14967771

Prediction of neuropsychological impairment in multiple sclerosis: comparison of conventional magnetic resonance imaging measures of atrophy and lesion burden.

Ralph H B Benedict1, Bianca Weinstock-Guttman, Inna Fishman, Jitendra Sharma, Christopher W Tjoa, Rohit Bakshi.   

Abstract

BACKGROUND: Cognition and magnetic resonance imaging correlations are well established in patients with multiple sclerosis (MS), but it is unclear whether lesion burden or atrophy accounts for most of the predictive variance. These indices have been directly compared in only a few studies. No such study included measurement of the third ventricle, which was strongly predictive of neuropsychological competence in the early literature. Furthermore, few studies accounted for the influence of age, premorbid intelligence, or depression.
OBJECTIVE: To determine if conventional measures of lesion burden or atrophy predict cognitive dysfunction in MS while accounting for age, premorbid intelligence, and depression.
METHODS: We studied 37 patients with MS and 27 controls matched according to demographic variables. Correlations between neuropsychological tests and the following magnetic resonance imaging indices were considered: T1 hypointense lesion volume, fluid-attenuated inversion recovery hyperintense lesion volume, third ventricle width, bicaudate ratio, and brain parenchymal fraction. Regression models predicting neuropsychological performance controlled for the effects of age, premorbid intelligence, and depression. We included only those tests discriminating patients with MS from controls.
RESULTS: In each regression model, third ventricle width was the sole magnetic resonance imaging measure retained. When this variable was removed from consideration, brain parenchymal fraction was retained in all analyses.
CONCLUSIONS: Brain atrophy accounts for more variance than lesion burden in predicting cognitive impairment in MS, and central atrophy in particular is strongly associated with neuropsychological morbidity. This finding may be explained in part by atrophy of the thalamus, a deep gray matter structure that mediates cognitive function via cortical and subcortical pathways. Enthusiasm for the clinical utility of third ventricle width is tempered by modest intraobserver and interobserver reliability.

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Mesh:

Year:  2004        PMID: 14967771     DOI: 10.1001/archneur.61.2.226

Source DB:  PubMed          Journal:  Arch Neurol        ISSN: 0003-9942


  100 in total

1.  Warmer outdoor temperature is associated with worse cognitive status in multiple sclerosis.

Authors:  Victoria M Leavitt; James F Sumowski; Nancy Chiaravalloti; John Deluca
Journal:  Neurology       Date:  2012-03-07       Impact factor: 9.910

2.  Whole-brain atrophy in multiple sclerosis measured by automated versus semiautomated MR imaging segmentation.

Authors:  Jitendra Sharma; Michael P Sanfilipo; Ralph H B Benedict; Bianca Weinstock-Guttman; Frederick E Munschauer; Rohit Bakshi
Journal:  AJNR Am J Neuroradiol       Date:  2004 Jun-Jul       Impact factor: 3.825

3.  Association between white matter microstructure, executive functions, and processing speed in older adults: the impact of vascular health.

Authors:  Heidi I L Jacobs; Elizabeth C Leritz; Victoria J Williams; Martin P J Van Boxtel; Wim van der Elst; Jelle Jolles; Frans R J Verhey; Regina E McGlinchey; William P Milberg; David H Salat
Journal:  Hum Brain Mapp       Date:  2011-09-23       Impact factor: 5.038

4.  Connectivity-based parcellation of the thalamus in multiple sclerosis and its implications for cognitive impairment: A multicenter study.

Authors:  Alvino Bisecco; Maria A Rocca; Elisabetta Pagani; Laura Mancini; Christian Enzinger; Antonio Gallo; Hugo Vrenken; Maria Laura Stromillo; Massimiliano Copetti; David L Thomas; Franz Fazekas; Gioacchino Tedeschi; Frederik Barkhof; Nicola De Stefano; Massimo Filippi
Journal:  Hum Brain Mapp       Date:  2015-04-14       Impact factor: 5.038

5.  Prediction of longitudinal brain atrophy in multiple sclerosis by gray matter magnetic resonance imaging T2 hypointensity.

Authors:  Robert A Bermel; Srinivas R Puli; Richard A Rudick; Bianca Weinstock-Guttman; Elizabeth Fisher; Frederick E Munschauer; Rohit Bakshi
Journal:  Arch Neurol       Date:  2005-09

6.  Cortical lesions in multiple sclerosis: combined postmortem MR imaging and histopathology.

Authors:  Jeroen J G Geurts; Lars Bö; Petra J W Pouwels; Jonas A Castelijns; Chris H Polman; Frederik Barkhof
Journal:  AJNR Am J Neuroradiol       Date:  2005-03       Impact factor: 3.825

Review 7.  Imaging of multiple sclerosis: role in neurotherapeutics.

Authors:  Rohit Bakshi; Alireza Minagar; Zeenat Jaisani; Jerry S Wolinsky
Journal:  NeuroRx       Date:  2005-04

Review 8.  Reliability and validity of neuropsychological screening and assessment strategies in MS.

Authors:  Ralph H B Benedict; Robert Zivadinov
Journal:  J Neurol       Date:  2007-05       Impact factor: 4.849

Review 9.  [Dementia as a primary symptom in late onset multiple sclerosis. Case series and review of the literature].

Authors:  T Leyhe; C Laske; G Buchkremer; H Wormstall; H Wiendl
Journal:  Nervenarzt       Date:  2005-06       Impact factor: 1.214

Review 10.  Grey matter lesions in MS: from histology to clinical implications.

Authors:  Massimiliano Calabrese; Alice Favaretto; Valeria Martini; Paolo Gallo
Journal:  Prion       Date:  2012-10-23       Impact factor: 3.931

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