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Literature DB >> 14752274

Neosis and its potential role in cancer development and chemoresistance.

Patrick M Navolanic¹, Shaw M Akula, James A McCubrey.

Abstract

Entities: Disease

Mesh：

Substances：

Year: 2004 PMID： 14752274 DOI： 10.4161/cbt.3.2.750

Source DB: PubMed Journal: Cancer Biol Ther ISSN： 1538-4047 Impact factor: 4.742

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Cited

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9 in total

1. Role of senescence and mitotic catastrophe in cancer therapy.

Authors: Richa Singh; Jasmine George; Yogeshwer Shukla
Journal: Cell Div Date: 2010-01-21 Impact factor: 5.130

Review 2. Therapeutic resistance and cancer recurrence mechanisms: Unfolding the story of tumour coming back.

Authors: Mohammad Javad Dehghan Esmatabadi; Babak Bakhshinejad; Fatemeh Movahedi Motlagh; Sadegh Babashah; Majid Sadeghizadeh
Journal: J Biosci Date: 2016-09 Impact factor: 1.826

Review 3. Docetaxel-induced polyploidization may underlie chemoresistance and disease relapse.

Authors: Angela Ogden; Padmashree C G Rida; Beatrice S Knudsen; Omer Kucuk; Ritu Aneja
Journal: Cancer Lett Date: 2015-07-13 Impact factor: 8.679

4. Genetic and pharmacological inhibition of acid ceramidase prevents asymmetric cell division by neosis.

Authors: Shai White-Gilbertson; Ping Lu; James S Norris; Christina Voelkel-Johnson
Journal: J Lipid Res Date: 2019-04-15 Impact factor: 5.922

Review 5. Significance of Wild-Type p53 Signaling in Suppressing Apoptosis in Response to Chemical Genotoxic Agents: Impact on Chemotherapy Outcome.

Authors: Razmik Mirzayans; Bonnie Andrais; Piyush Kumar; David Murray
Journal: Int J Mol Sci Date: 2017-04-28 Impact factor: 5.923

Neosis and its potential role in cancer development and chemoresistance.

1. Role of senescence and mitotic catastrophe in cancer therapy.

Review 2. Therapeutic resistance and cancer recurrence mechanisms: Unfolding the story of tumour coming back.

Review 3. Docetaxel-induced polyploidization may underlie chemoresistance and disease relapse.

4. Genetic and pharmacological inhibition of acid ceramidase prevents asymmetric cell division by neosis.

Review 5. Significance of Wild-Type p53 Signaling in Suppressing Apoptosis in Response to Chemical Genotoxic Agents: Impact on Chemotherapy Outcome.

6. Multinucleated polyploidy drives resistance to Docetaxel chemotherapy in prostate cancer.

7. A Fraction of CD133+ CNE2 Cells Is Made of Giant Cancer Cells with Morphological Evidence of Asymmetric Mitosis.

Review 8. Ionizing radiation-induced responses in human cells with differing TP53 status.

9. Irradiation-induced polyploid giant cancer cells are involved in tumor cell repopulation via neosis.