Literature DB >> 14736962

Expression of human nephrin mRNA in diabetic nephropathy.

Masao Toyoda1, Daisuke Suzuki, Tomoya Umezono, Goro Uehara, Mayumi Maruyama, Masashi Honma, Takako Sakai, Hideto Sakai.   

Abstract

BACKGROUND: Diabetic nephropathy (DN) is associated with functional changes in the filtration barrier, and microalbuminuria is a strong predictor of the development of overt DN. Nephrin is a novel podocyte-specific protein which localizes at the slit diaphragm. This study examines the expression of nephrin mRNA in the kidneys of type 2 diabetics with DN.
METHODS: Renal tissues were obtained from 13 type 2 diabetics with DN. We also examined samples from five patients with minimal change nephrotic syndrome (MCNS) and five normal kidneys (normals) as control. The severity of DN was classified into two grades based on histopathological findings. DN grade 1 (DN1 = seven patients) presented mild mesangial expansion, and DN grade 2 (DN2 = six patients) moderate mesangial expansion. Nephrin mRNA was quantitated and localized by in situ hybridization.
RESULTS: Cells positive for nephrin mRNA were detected exclusively in glomerular epithelial cells. The percentage of cells positive for nephrin mRNA in DN2 was significantly lower than in MCNS and normal kidneys. Furthermore, there was an inverse correlation between the percentage of cells positive for nephrin mRNA and extent of proteinuria.
CONCLUSION: The low expression of nephrin mRNA may be closely linked to development and/or progression of proteinuria in human diabetic nephropathy.

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Year:  2004        PMID: 14736962     DOI: 10.1093/ndt/gfg545

Source DB:  PubMed          Journal:  Nephrol Dial Transplant        ISSN: 0931-0509            Impact factor:   5.992


  22 in total

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4.  Nephrin Preserves Podocyte Viability and Glomerular Structure and Function in Adult Kidneys.

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Review 5.  Gene expression in diabetic nephropathy.

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8.  Hyperoside reduces albuminuria in diabetic nephropathy at the early stage through ameliorating renal damage and podocyte injury.

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9.  BMP signaling and podocyte markers are decreased in human diabetic nephropathy in association with CTGF overexpression.

Authors:  Tamara Turk; Jan Willem Leeuwis; Julia Gray; Suzy V Torti; Karen M Lyons; Tri Q Nguyen; Roel Goldschmeding
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10.  Regulation of Neph3 gene in podocytes--key roles of transcription factors NF-kappaB and Sp1.

Authors:  Mervi Ristola; Satu Arpiainen; Moin A Saleem; Peter W Mathieson; Gavin I Welsh; Sanna Lehtonen; Harry Holthöfer
Journal:  BMC Mol Biol       Date:  2009-08-24       Impact factor: 2.946

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