Literature DB >> 14662791

Preservation of spermatogenesis in spinal cord injured rats with exogenous testosterone. Relationship with serum testosterone levels and cellular localization of cAMP responsive element modulator.

Hosea F S Huang1, Shulun Wang, Carlos A Molina, John E Ottenweller.   

Abstract

UNLABELLED: The current experiment examined the effects of exogenous testosterone (T) on spermatogenesis in rats with spinal cord injury (SCI) and their relationship with the cellular distribution of a cyclic AMP-responsive element modulator (CREM) in testicular cells. Implantation of T-filled Silastic capsules (TCs, 1-20 cm) resulted in dose-dependent, biphasic changes in testicular T levels and spermatogenesis in SCI rats. However, dose responsiveness of spermatogenesis to exogenous T in SCI rats differed from that in sham control rats. Specifically, implantation of 2-cm TCs enhanced the effects of SCI on spermatogenesis, resulting in total regression of the seminiferous epithelium. Although 3-cm TCs maintained complete spermatogenesis in sham control rats, this regimen failed to support complete spermatogenesis in SCI rats. Although complete spermatogenesis was maintained in SCI rats given 5-20-cm TC implants, various abnormalities persisted. Cellular distribution of CREM remained normal in SCI rats but was altered in those SCI rats that received 3- or 5-cm TC implants. Such effects were associated with reduced CREM proteins in testicular tissues. These results were consistent with altered cAMP signaling and its regulation in testicular cells after SCI and provided possible mechanistic explanations for the effects of SCI on spermatogenesis.
CONCLUSION: SCI resulted in changes in the responsiveness of spermatogenesis to exogenous T. These effects were associated with altered cAMP/CREM signaling in testicular cells. Further studies, including a study of the relationship between serum T levels and normalcy of sperm functions and the role of neural-endocrine interactions in mediating the effects of SCI on spermatogenesis and sperm function, are needed so that therapeutic regimens can be designed for clinical use.

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Year:  2004        PMID: 14662791     DOI: 10.1002/j.1939-4640.2004.tb02763.x

Source DB:  PubMed          Journal:  J Androl        ISSN: 0196-3635


  4 in total

1.  The concentration of androgen receptor and protein kinase A in male chicken following the administration of a combination of the epididymis and testicular extracts.

Authors:  Muslim Akmal; Gholib Gholib; Mustafa Kamal Nasution; Sri Wahyuni; Rinidar Rinidar; Dian Masyitha; M Aman Yaman
Journal:  Vet World       Date:  2020-08-15

2.  Spinal cord injury causes sustained disruption of the blood-testis barrier in the rat.

Authors:  Jennifer N Dulin; Meredith L Moore; Kevin W Gates; Joanna H Queen; Raymond J Grill
Journal:  PLoS One       Date:  2011-01-26       Impact factor: 3.240

3.  Testosterone Reduces Spinal Cord Injury-Induced Effects on Male Reproduction by Preventing CADM1 Defect.

Authors:  Hamid Choobineh; Mahsa Kazemi; Mohammad Ali Sadighi Gilani; Tahereh Heydari; Saeed Shokri; Mahshid Bazrafkan; Gholamreza Hassanzadeh
Journal:  Cell J       Date:  2018-03-18       Impact factor: 2.479

4.  The concentration of testosterone, pituitary adenylate cyclase-activating polypeptide, and protamine 1 in the serum of male chicken following administration of epididymis and testicular extracts and their combination.

Authors:  Muslim Akmal; Gholib Gholib; Rinidar Rinidar; Fitriani Fitriani; T Zahrial Helmi; Sugito Sugito; M Isa; Nurliana Nurliana; Sri Wahyuni; Dasrul Dasrul; M Aman Yaman
Journal:  Vet World       Date:  2019-07-25
  4 in total

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