Literature DB >> 14602772

"Incipient" primary hyperparathyroidism: a "forme fruste" of an old disease.

Shonni J Silverberg1, John P Bilezikian.   

Abstract

Although primary hyperparathyroidism today is often a relatively asymptomatic disease, it has distinct biochemical and skeletal features. These features are present at diagnosis and are generally stable over time, leading to the theory of a biphasic disease course in which alterations occur during a preclinical phase. Measurement of calciotropic hormones in individuals undergoing skeletal evaluation has led to the identification of normocalcemic individuals with elevated PTH levels. We hypothesize that these patients represent the earliest manifestations of primary hyperparathyroidism Twenty-two patients had hyperparathyroidism (94 +/- 29 pg/ml) and normal corrected serum calcium levels (2.40 +/- 0.02 mmol/liter). No secondary causes of hyperparathyroidism were found. PTH levels did not correlate with urinary calcium concentration, renal function, vitamin D concentrations, or bone density. The relationship between PTH and serum calcium (regression slope, +0.004) was identical in normocalcemic and hypercalcemic hyperparathyroid patients. Preferential cortical bone loss, characteristic of patients with primary hyperparathyroidism, was not seen (T-score: spine, -1.6; hip, -1.8; distal one-third radius, -1.3). In up to 12 months of observation, three patients have developed hypercalcemia, and one has had two adenomas removed. These patients with elevated PTH levels in the absence of hypercalcemia may provide a window into this previously unrecognized stage of the disease and permit investigators to track its evolution in ways that have not heretofore been possible.

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Year:  2003        PMID: 14602772     DOI: 10.1210/jc.2003-031014

Source DB:  PubMed          Journal:  J Clin Endocrinol Metab        ISSN: 0021-972X            Impact factor:   5.958


  39 in total

Review 1.  Bone strength in primary hyperparathyroidism.

Authors:  John P Bilezikian
Journal:  Osteoporos Int       Date:  2003-08-29       Impact factor: 4.507

2.  Glucose intolerance and primary hyperparathyroidism: an unresolved relationship.

Authors:  Mishaela R Rubin; Shonni J Silverberg
Journal:  Endocrine       Date:  2012-10       Impact factor: 3.633

3.  Primary Hyperparathyroidism: A Tale of Two Cities Revisited - New York and Shanghai.

Authors:  Jian-Min Liu; Natalie E Cusano; Barbara C Silva; Lin Zhao; Xiao-Yan He; Bei Tao; Li-Hao Sun; Hong-Yan Zhao; Wen-Wei Fan; Megan E Romano; Guang Ning; John P Bilezikian
Journal:  Bone Res       Date:  2013-06-28       Impact factor: 13.567

Review 4.  Lessons from second- and third-generation parathyroid hormone assays in primary hyperparathyroidism.

Authors:  J C Souberbielle; P Boudou; C Cormier
Journal:  J Endocrinol Invest       Date:  2008-05       Impact factor: 4.256

Review 5.  Mild primary hyperparathyroidism: a literature review.

Authors:  Megan K Applewhite; David F Schneider
Journal:  Oncologist       Date:  2014-07-25

6.  Normocalcemic primary hyperparathyroidism in clinical practice: an indolent condition or a silent threat?

Authors:  Thyciara Fontenele Marques; Renata Vasconcelos; Erik Diniz; Daniela Rêgo; Luiz Griz; Francisco Bandeira
Journal:  Arq Bras Endocrinol Metabol       Date:  2011-06

7.  Surgery of parathyroid.

Authors:  Sai Krishna Vittal; V Sai Vishnupriya; V Sucharitha; S Vittal
Journal:  Indian J Surg       Date:  2010-02-05       Impact factor: 0.656

Review 8.  Normocalcemic primary hyperparathyroidism.

Authors:  Natalie E Cusano; Shonni J Silverberg; John P Bilezikian
Journal:  J Clin Densitom       Date:  2013 Jan-Mar       Impact factor: 2.617

9.  Primary hyperparathyroidism associated with hypocalcemia in a patient presenting with kidney disease.

Authors:  Sylvie Dusilova Sulkova; Jiri Horacek; Pavel Zivny; Pavla Rehorkova; Miloslav Podhola; Mirko Kadlec; Ctibor Povysil
Journal:  Int Urol Nephrol       Date:  2010-05-07       Impact factor: 2.370

10.  A novel loss-of-function mutation, Gln459Arg, of the calcium-sensing receptor gene associated with apparent autosomal recessive inheritance of familial hypocalciuric hypercalcemia.

Authors:  Steven A Lietman; Yardena Tenenbaum-Rakover; Tjin Shing Jap; Wu Yi-Chi; Yang De-Ming; Changlin Ding; Najat Kussiny; Michael A Levine
Journal:  J Clin Endocrinol Metab       Date:  2009-09-29       Impact factor: 5.958

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