Literature DB >> 14515119

On the implications of polyclonal B cell activation.

Arthur M Silverstein, Noel R Rose.   

Abstract

Mesh：

Year:  2003        PMID： 14515119     DOI： 10.1038/ni1003-931

Source DB:  PubMed          Journal:  Nat Immunol        ISSN： 1529-2908            Impact factor:   25.606


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  5 in total

1.  Autoantigen, innate immunity, and T cells cooperate to break B cell tolerance during bacterial infection.

Authors:  Pauline Soulas; Anne Woods; Benoit Jaulhac; Anne-Marie Knapp; Jean-Louis Pasquali; Thierry Martin; Anne-Sophie Korganow
Journal:  J Clin Invest       Date:  2005-07-21       Impact factor: 14.808

2.  Influenza virus-induced type I interferon leads to polyclonal B-cell activation but does not break down B-cell tolerance.

Authors:  Anne Woods; Fanny Monneaux; Pauline Soulas-Sprauel; Sylviane Muller; Thierry Martin; Anne-Sophie Korganow; Jean-Louis Pasquali
Journal:  J Virol       Date:  2007-09-12       Impact factor: 5.103

3.  MyD88 negatively controls hypergammaglobulinemia with autoantibody production during bacterial infection.

Authors:  Anne Woods; Pauline Soulas-Sprauel; Benoit Jaulhac; Bérénice Arditi; Anne-Marie Knapp; Jean-Louis Pasquali; Anne-Sophie Korganow; Thierry Martin
Journal:  Infect Immun       Date:  2008-01-28       Impact factor: 3.441

4.  Toll-like receptor agonists synergize with CD40L to induce either proliferation or plasma cell differentiation of mouse B cells.

Authors:  Emmanuelle Boeglin; Cristian R Smulski; Susana Brun; Sara Milosevic; Pascal Schneider; Sylvie Fournel
Journal:  PLoS One       Date:  2011-10-03       Impact factor: 3.240

Review 5.  Immune Response Regulation by Antigen Receptors' Clone-Specific Nonself Parts.

Authors:  Hilmar Lemke
Journal:  Front Immunol       Date:  2018-06-25       Impact factor: 7.561
  5 in total

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