Literature DB >> 1424691

Increased gut permeability early after burns correlates with the extent of burn injury.

C M Ryan1, M L Yarmush, J F Burke, R G Tompkins.   

Abstract

OBJECTIVE: To determine if increased gut permeability within 48 hrs after burn injury correlates with the extent of injury, before sepsis and pulmonary disorders have complicated the clinical course.
DESIGN: Nonrandomized, controlled study. PATIENTS: Consecutive patients admitted with burn injuries on > 20% of body surface area.
INTERVENTIONS: Intestinal absorption and renal excretion of polyethylene glycol 3350 was used as the macromolecule to determine gut permeability; polyethylene glycol 400 intestinal absorption was used as an internal control for abnormal motility and malabsorption. Polyethylene glycol 3350 (40 g) and polyethylene glycol 400 (5 g) were administered enterally.
MEASUREMENTS AND MAIN RESULTS: Gut permeability was significantly increased early after the injury. The patients excreted 0.56 +/- 0.34% (n = 11) of polyethylene glycol 3350, compared with the amount (0.12 +/- 0.04%) (p < .05) previously reported in normal volunteers. There was no significant difference in the excretion of polyethylene glycol 400 in the patients (27.0 +/- 4.6%, n = 11) vs. the normal volunteers previously reported (26.3 +/- 5.1%, n = 12), suggesting normal intestinal motility and absorption. The percentage of excretion of polyethylene glycol 3350 correlated with the percentage body surface burned; patients with smaller injuries excreted 0.32 +/- 0.17% (n = 6), which was greater than normal and less than those values from patients with larger injuries, 0.84 +/- 0.25% (n = 5) (p < .001 by Turkey test).
CONCLUSIONS: Using our newly developed method to separate and quantify polyethylene glycols in urine, gut permeability was found to be increased early after burn injury, which confirms a previous study using lactulose as the permeability probe. Furthermore, this increased gut permeability to polyethylene glycol 3350 correlated with the extent of the burn injury.

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Year:  1992        PMID: 1424691     DOI: 10.1097/00003246-199211000-00005

Source DB:  PubMed          Journal:  Crit Care Med        ISSN: 0090-3493            Impact factor:   7.598


  13 in total

1.  Protein losing enteropathy in critically ill adult patients with burns: a preliminary report.

Authors:  Balasubramanian Venkatesh; Jenny Gough; David R Ralston; Michael Muller; Stuart Pegg
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Review 2.  Use of plasma procalcitonin levels as an adjunct to clinical microbiology.

Authors:  David N Gilbert
Journal:  J Clin Microbiol       Date:  2010-04-26       Impact factor: 5.948

3.  Efficacy of enteral glutamine supplementation in patients with severe and predicted severe acute pancreatitis- A randomized controlled trial.

Authors:  Madhulika Arutla; M Raghunath; G Deepika; Aparna Jakkampudi; H V V Murthy; G V Rao; D Nageshwar Reddy; Rupjyoti Talukdar
Journal:  Indian J Gastroenterol       Date:  2019-10-14

Review 4.  Procalcitonin in sepsis and systemic inflammation: a harmful biomarker and a therapeutic target.

Authors:  Kenneth L Becker; Richard Snider; Eric S Nylen
Journal:  Br J Pharmacol       Date:  2009-11-27       Impact factor: 8.739

Review 5.  Selective digestive decontamination (SDD) as a tool in the management of bacterial translocation following major burns.

Authors:  Y A Aboelatta; A M Abd-Elsalam; A H Omar; M M Abdelaal; A M Farid
Journal:  Ann Burns Fire Disasters       Date:  2013-12-31

6.  Lactobacillus plantarum 299v inhibits Escherichia coli-induced intestinal permeability.

Authors:  Peter Mangell; Pernilla Nejdfors; Mei Wang; Siv Ahrné; Bjorn Weström; Henrik Thorlacius; Bengt Jeppsson
Journal:  Dig Dis Sci       Date:  2002-03       Impact factor: 3.199

7.  Burn and starvation increase programmed cell death in small bowel epithelial cells.

Authors:  M G Jeschke; M A Debroy; S E Wolf; S Rajaraman; J C Thompson
Journal:  Dig Dis Sci       Date:  2000-02       Impact factor: 3.199

8.  Insulin-like growth factor-1 levels contribute to the development of bacterial translocation in sepsis.

Authors:  Gary W Hunninghake; Kevin C Doerschug; Amanda B Nymon; Gregory A Schmidt; David K Meyerholz; Alix Ashare
Journal:  Am J Respir Crit Care Med       Date:  2010-04-22       Impact factor: 21.405

9.  Burn injury-induced alterations in wound inflammation and healing are associated with suppressed hypoxia inducible factor-1alpha expression.

Authors:  Martin G Schwacha; Eike Nickel; TanJanika Daniel
Journal:  Mol Med       Date:  2008 Sep-Oct       Impact factor: 6.354

Review 10.  Clinical pharmacokinetics in patients with burns.

Authors:  U Jaehde; F Sörgel
Journal:  Clin Pharmacokinet       Date:  1995-07       Impact factor: 6.447

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