Literature DB >> 1401310

The focal facial dermal dysplasias: report of a kindred and a proposed new classification.

D C Kowalski1, N A Fenske.   

Abstract

BACKGROUND: The focal facial dermal dysplasias (FFDD) are a genetically heterogeneous group of disorders characterized by congenital bilateral scarlike facial lesions, with or without associated facial anomalies. The cases have been reported under various names; thus the nosology is confusing and unclear.
OBJECTIVE: Our purposes were to report our kindred, clearly delineate the various types of FFDD reported, and propose a new simplified classification.
METHODS: The clinical and histologic changes were examined and genealogy determined for our kindred. The medical literature was reviewed and the reported cases reexamined and categorized according to their clinical features and inheritance patterns.
RESULTS: We determined that there are three distinct varieties of FFDD: type I, autosomal dominant FFDD; type II, autosomal recessive FFDD; and type III, FFDD with other facial features.
CONCLUSION: We propose a new classification and provide evidence for three distinct varieties of FFDD: type I, autosomal dominant FFDD; type II, autosomal recessive FFDD; and type III, FFDD with other facial features (Setleis syndrome). Our kindred represents type II.

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Mesh:

Year:  1992        PMID: 1401310     DOI: 10.1016/0190-9622(92)70225-5

Source DB:  PubMed          Journal:  J Am Acad Dermatol        ISSN: 0190-9622            Impact factor:   11.527


  4 in total

1.  Focal facial dermal dysplasia, type IV, is caused by mutations in CYP26C1.

Authors:  Anne M Slavotinek; Pavni Mehrotra; Irina Nazarenko; Paul Ling-Fung Tang; Richard Lao; Don Cameron; Ben Li; Catherine Chu; Chris Chou; Ann L Marqueling; Mani Yahyavi; Kelly Cordoro; Ilona Frieden; Tom Glaser; Trine Prescott; Marie-Anne Morren; Koen Devriendt; Pui-yan Kwok; Martin Petkovich; Robert J Desnick
Journal:  Hum Mol Genet       Date:  2012-11-16       Impact factor: 6.150

2.  Focal facial dermal dysplasia type 4: identification of novel CYP26C1 mutations in unrelated patients.

Authors:  Beom Hee Lee; Fanny Morice-Picard; Franck Boralevi; Brenden Chen; Robert J Desnick
Journal:  J Hum Genet       Date:  2017-12-20       Impact factor: 3.172

3.  Setleis syndrome: clinical, molecular and structural studies of the first TWIST2 missense mutation.

Authors:  R Ozgur Rosti; Z Oya Uyguner; Irina Nazarenko; Mehmet Bekerecioglu; Carmen L Cadilla; Hilal Ozgur; Beom Hee Lee; Aneel K Aggarwal; Sacide Pehlivan; Robert J Desnick
Journal:  Clin Genet       Date:  2014-12-11       Impact factor: 4.438

4.  Homozygous nonsense mutations in TWIST2 cause Setleis syndrome.

Authors:  Turgut Tukel; Drazen Šošić; Lihadh I Al-Gazali; Mónica Erazo; Jose Casasnovas; Hector L Franco; James A Richardson; Eric N Olson; Carmen L Cadilla; Robert J Desnick
Journal:  Am J Hum Genet       Date:  2010-08-13       Impact factor: 11.025

  4 in total

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