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Literature DB >> 13714542

Mechanism of natural and acquired resistance to methyl-bis-(beta-chlorethyl)-amine N-oxide in ascites tumors.

I HIRONO.

Abstract

Entities: Chemical Disease

Keywords: NITROGEN MUSTARDS/pharmacology

Mesh：

Substances：

Year: 1961 PMID： 13714542

Source DB: PubMed Journal: Gan ISSN： 0016-450X

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6 in total

1. A synthetic lethal drug combination mimics glucose deprivation-induced cancer cell death in the presence of glucose.

Authors: James H Joly; Alireza Delfarah; Philip S Phung; Sydney Parrish; Nicholas A Graham
Journal: J Biol Chem Date: 2019-12-30 Impact factor: 5.157

2. Deubiquitinases Maintain Protein Homeostasis and Survival of Cancer Cells upon Glutathione Depletion.

Authors: Isaac S Harris; Jennifer E Endress; Jonathan L Coloff; Laura M Selfors; Samuel K McBrayer; Jennifer M Rosenbluth; Nobuaki Takahashi; Sabin Dhakal; Vidyasagar Koduri; Matthew G Oser; Nathan J Schauer; Laura M Doherty; Andrew L Hong; Yun Pyo Kang; Scott T Younger; John G Doench; William C Hahn; Sara J Buhrlage; Gina M DeNicola; William G Kaelin; Joan S Brugge
Journal: Cell Metab Date: 2019-02-21 Impact factor: 27.287

3. Differential properties of cisplatin and tetraplatin with respect to cytotoxicity and perturbation of cellular glutathione levels.

Authors: M R Müller; K A Wright; P R Twentyman
Journal: Cancer Chemother Pharmacol Date: 1991 Impact factor: 3.333

Mechanism of natural and acquired resistance to methyl-bis-(beta-chlorethyl)-amine N-oxide in ascites tumors.

1. A synthetic lethal drug combination mimics glucose deprivation-induced cancer cell death in the presence of glucose.

2. Deubiquitinases Maintain Protein Homeostasis and Survival of Cancer Cells upon Glutathione Depletion.

3. Differential properties of cisplatin and tetraplatin with respect to cytotoxicity and perturbation of cellular glutathione levels.

Review 4. Importance of the trans-sulfuration pathway in cancer prevention and promotion.

Review 5. ROS Pleiotropy in Melanoma and Local Therapy with Physical Modalities.

6. Combined effect of buthionine sulfoximine and cyclophosphamide upon murine tumours and bone marrow.