Literature DB >> 12970634

Antenatal manifestations of mitochondrial respiratory chain deficiency.

Jürgen-Christoph von Kleist-Retzow1, Valérie Cormier-Daire, Géraldine Viot, Alice Goldenberg, Becky Mardach, Jeanne Amiel, Philippe Saada, Yves Dumez, Francis Brunelle, Jean-Marie Saudubray, Dominique Chrétien, Agnès Rötig, Pierre Rustin, Arnold Munnich, Pascale De Lonlay.   

Abstract

OBJECTIVE: To review the antenatal manifestations of disorders of oxidative phosphorylation. STUDY
DESIGN: A total of 300 cases of proven respiratory chain enzyme deficiency were retrospectively reviewed for fetal development, based on course and duration of pregnancy, antenatal ultrasonography and birth weight, length, and head circumference. Particular attention was given to fetal movements, oligo/hydramnios, fetal cardiac rhythm, fetal heart ultrasound, and ultrasonography/echo Doppler signs of brain, facial, trunk, limb, and organ anomalies.
RESULTS: Retrospective analyses detected low birth weight (<3rd percentile for gestational age) in 22.7% of cases (68/300, P<.000001). Intrauterine growth retardation was either isolated (48/300, 16%) or associated with otherwise unexplained anomalies (20/300, 6.7%, P<.0001). Antenatal anomalies were usually multiple and involved several organs sharing no common function or embryologic origin. They included polyhydramnios (6/20), oligoamnios (2/20), arthrogryposis (1/20), decreased fetal movements (1/20), ventricular septal defects (2/20), hypertrophic cardiomyopathy (4/20), cardiac rhythm anomalies (4/20), hydronephrosis (3/20), vertebral abnormalities, anal atresia, cardiac abnormalities, tracheoesophageal fistula/atresia, renal agenesis and dysplasia, and limb defects (VACTERL) association (2/20), and a complex gastrointestinal malformation (1/20).
CONCLUSIONS: Although a number of metabolic diseases undergo a symptom-free period, respiratory chain deficiency may have an early antenatal expression, presumably related to the time course of the disease gene expression in the embryofetal period. The mechanism triggering malformations is unknown and may include decreased ATP formation and/or an alteration of apoptotic events controlled by the mitochondria.

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Mesh:

Year:  2003        PMID: 12970634     DOI: 10.1067/S0022-3476(03)00130-6

Source DB:  PubMed          Journal:  J Pediatr        ISSN: 0022-3476            Impact factor:   4.406


  31 in total

Review 1.  Antenatal manifestations of inborn errors of metabolism: biological diagnosis.

Authors:  Christine Vianey-Saban; Cécile Acquaviva; David Cheillan; Sophie Collardeau-Frachon; Laurent Guibaud; Cécile Pagan; Magali Pettazzoni; Monique Piraud; Antonin Lamazière; Roseline Froissart
Journal:  J Inherit Metab Dis       Date:  2016-07-08       Impact factor: 4.982

2.  Conventional MRI and MR spectroscopy in nonclassical mitochondrial disease: report of three patients with mitochondrial DNA deletion.

Authors:  Leonardo Vedolin; Carolina Fischinger Moura de Souza; Rogério Schwark Silveira; Bianca Cunha Lopes; Leticia Saldanha Laybauer; Maria Luiza Saraiva Pereira; Roberto Giugliani
Journal:  Childs Nerv Syst       Date:  2006-03-22       Impact factor: 1.475

3.  Mitochondrial Factors and VACTERL Association-Related Congenital Malformations.

Authors:  S Siebel; B D Solomon
Journal:  Mol Syndromol       Date:  2013-02

Review 4.  Antenatal manifestations of inborn errors of metabolism: autopsy findings suggestive of a metabolic disorder.

Authors:  Sophie Collardeau-Frachon; Marie-Pierre Cordier; Massimiliano Rossi; Laurent Guibaud; Christine Vianey-Saban
Journal:  J Inherit Metab Dis       Date:  2016-04-22       Impact factor: 4.982

5.  Maternal Multiple Micronutrient Supplementation Stabilizes Mitochondrial DNA Copy Number in Pregnant Women in Lombok, Indonesia.

Authors:  Lidwina Priliani; Elizabeth L Prado; Restuadi Restuadi; Diana E Waturangi; Anuraj H Shankar; Safarina G Malik
Journal:  J Nutr       Date:  2019-08-01       Impact factor: 4.798

Review 6.  Antenatal manifestations of inborn errors of metabolism: prenatal imaging findings.

Authors:  Laurent Guibaud; Sophie Collardeau-Frachon; Audrey Lacalm; Mona Massoud; Massimiliano Rossi; Marie Pierre Cordier; Christine Vianey-Saban
Journal:  J Inherit Metab Dis       Date:  2016-11-16       Impact factor: 4.982

7.  Analysis of component findings in 79 patients diagnosed with VACTERL association.

Authors:  Benjamin D Solomon; Daniel E Pineda-Alvarez; Manu S Raam; Sophia M Bous; Amelia A Keaton; Jorge I Vélez; Derek A T Cummings
Journal:  Am J Med Genet A       Date:  2010-09       Impact factor: 2.802

8.  Congenital cardiomyopathy and pulmonary hypertension: another fatal variant of cytochrome-c oxidase deficiency.

Authors:  C P Venditti; M C Harris; D Huff; I Peterside; D Munson; H S Weber; J Rome; E M Kaye; S Shanske; S Sacconi; S Tay; S DiMauro; G T Berry
Journal:  J Inherit Metab Dis       Date:  2004       Impact factor: 4.982

9.  Survival among children with "Lethal" congenital contracture syndrome 11 caused by novel mutations in the gliomedin gene (GLDN).

Authors:  Jennifer A Wambach; Georg M Stettner; Tobias B Haack; Karin Writzl; Andreja Škofljanec; Aleš Maver; Francina Munell; Stephan Ossowski; Mattia Bosio; Daniel J Wegner; Marwan Shinawi; Dustin Baldridge; Bader Alhaddad; Tim M Strom; Dorothy K Grange; Ekkehard Wilichowski; Robin Troxell; James Collins; Barbara B Warner; Robert E Schmidt; Alan Pestronk; F Sessions Cole; Robert Steinfeld
Journal:  Hum Mutat       Date:  2017-08-17       Impact factor: 4.878

10.  Complex I function is defective in complex IV-deficient Caenorhabditis elegans.

Authors:  Wichit Suthammarak; Yu-Ying Yang; Phil G Morgan; Margaret M Sedensky
Journal:  J Biol Chem       Date:  2008-12-12       Impact factor: 5.157

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