Literature DB >> 12922124

Cross-protection between distantly related spotted fever group rickettsiae.

Hui-min Feng1, David H Walker.   

Abstract

Two excellent C3H/HeN mouse models of spotted fever rickettsioses caused by the distantly related organisms, Rickettsia conorii and Rickettsia australis, were utilized to evaluate the possibility of the stimulation of broad cross-protective immunity. Sublethal infection stimulated complete immunity, that is absence of disease, after challenge with a dose of the heterologous Rickettsia that uniformly killed naïve mice. In contrast, heterologous immune sera did not protect mice against a lethal dose (two LD50) of rickettsiae in the mouse toxicity neutralization assay, the standard method for evaluation of rickettsial vaccine potency. These observations suggest that development of a broadly protective vaccine against spotted fever group (SFG) rickettsiae is feasible, and the results indicate that mouse toxicity neutralization is an inappropriate method for evaluation of candidate vaccines.

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Year:  2003        PMID: 12922124     DOI: 10.1016/s0264-410x(03)00301-3

Source DB:  PubMed          Journal:  Vaccine        ISSN: 0264-410X            Impact factor:   3.641


  16 in total

Review 1.  Rickettsia australis and Queensland Tick Typhus: A Rickettsial Spotted Fever Group Infection in Australia.

Authors:  Adam Stewart; Mark Armstrong; Stephen Graves; Krispin Hajkowicz
Journal:  Am J Trop Med Hyg       Date:  2017-07       Impact factor: 2.345

2.  Effects of homologous and heterologous immunization on the reservoir competence of domestic dogs for Rickettsia conorii (israelensis).

Authors:  M L Levin; G E Zemtsova; M Montgomery; L F Killmaster
Journal:  Ticks Tick Borne Dis       Date:  2013-11-05       Impact factor: 3.744

3.  OmpA-mediated rickettsial adherence to and invasion of human endothelial cells is dependent upon interaction with α2β1 integrin.

Authors:  Robert D Hillman; Yasmine M Baktash; Juan J Martinez
Journal:  Cell Microbiol       Date:  2012-12-06       Impact factor: 3.715

4.  MyD88 Mediates Instructive Signaling in Dendritic Cells and Protective Inflammatory Response during Rickettsial Infection.

Authors:  Jeremy Bechelli; Claire Smalley; Xuemei Zhao; Barbara Judy; Patricia Valdes; David H Walker; Rong Fang
Journal:  Infect Immun       Date:  2016-03-24       Impact factor: 3.441

5.  Failure of a heterologous recombinant Sca5/OmpB protein-based vaccine to elicit effective protective immunity against Rickettsia rickettsii infections in C3H/HeN mice.

Authors:  Sean P Riley; Marissa M Cardwell; Yvonne G Y Chan; Ludovic Pruneau; Fabio Del Piero; Juan J Martinez
Journal:  Pathog Dis       Date:  2015-10-29       Impact factor: 3.166

6.  Contribution of NK cells to the innate phase of host protection against an intracellular bacterium targeting systemic endothelium.

Authors:  Rong Fang; Nahed Ismail; David H Walker
Journal:  Am J Pathol       Date:  2012-05-19       Impact factor: 4.307

7.  Phenotype of the anti-Rickettsia CD8(+) T cell response suggests cellular correlates of protection for the assessment of novel antigens.

Authors:  Erika Caro-Gomez; Michal Gazi; Maria A Cespedes; Yenny Goez; Bruno Teixeira; Gustavo Valbuena
Journal:  Vaccine       Date:  2014-07-18       Impact factor: 3.641

Review 8.  The realities of biodefense vaccines against Rickettsia.

Authors:  David H Walker
Journal:  Vaccine       Date:  2009-11-05       Impact factor: 3.641

9.  Rickettsial outer-membrane protein B (rOmpB) mediates bacterial invasion through Ku70 in an actin, c-Cbl, clathrin and caveolin 2-dependent manner.

Authors:  Yvonne G Y Chan; Marissa M Cardwell; Timothy M Hermanas; Tsuneo Uchiyama; Juan J Martinez
Journal:  Cell Microbiol       Date:  2009-01-07       Impact factor: 3.715

10.  Effect of antibody on the rickettsia-host cell interaction.

Authors:  Hui-Min Feng; Ted Whitworth; Vsevolod Popov; David H Walker
Journal:  Infect Immun       Date:  2004-06       Impact factor: 3.441

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