Literature DB >> 12836637

Hearts of this ILK rely on TNNI3K, a MAPKKK that regulated TNNI3.

Friedrich C Luft1.   

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Year:  2003        PMID： 12836637     DOI： 10.1007/s00109-003-0436-9

Source DB:  PubMed          Journal:  J Mol Med (Berl)        ISSN： 0946-2716            Impact factor:   4.599


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  2 in total

Review 1.  The genetic basis for cardiomyopathy: from mutation identification to mechanistic paradigms.

Authors:  J G Seidman; C Seidman
Journal:  Cell       Date:  2001-02-23       Impact factor: 41.582

2.  Cloning and characterization of a novel cardiac-specific kinase that interacts specifically with cardiac troponin I.

Authors:  Yong Zhao; Xian-Min Meng; Ying-Jie Wei; Xiu-Wen Zhao; Dong-Qing Liu; Hui-Qing Cao; Choong-Chin Liew; Jin-Feng Ding
Journal:  J Mol Med (Berl)       Date:  2003-04-30       Impact factor: 4.599
  2 in total
  2 in total

1.  Overexpression of TNNI3K, a cardiac-specific MAPKKK, promotes cardiac dysfunction.

Authors:  Hao Tang; Kunhong Xiao; Lan Mao; Howard A Rockman; Douglas A Marchuk
Journal:  J Mol Cell Cardiol       Date:  2012-10-16       Impact factor: 5.000

Review 2.  The Diverse Roles of TNNI3K in Cardiac Disease and Potential for Treatment.

Authors:  Caroline Pham; Noelia Muñoz-Martín; Elisabeth M Lodder
Journal:  Int J Mol Sci       Date:  2021-06-15       Impact factor: 5.923
  2 in total

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