Literature DB >> 12827053

Inhibition of corneal neovascularization by genetic ablation of CCR2.

Balamurali K Ambati1, Antonia M Joussen, William A Kuziel, Anthony P Adamis, Jayakrishna Ambati.   

Abstract

PURPOSE: To determine if genetic ablation of the chemokine receptor CCR2 (involved in leukocyte and endothelial chemotaxis) inhibits the development of corneal neovascularization.
METHODS: Wild-type C57BL/6J mice, as well as species-specific counterparts with targeted homozygous disruption of the CCR2, underwent chemical and mechanical denudation of corneal and limbal epithelium. Corneas were harvested 2 weeks after injury. Neovascularization was quantified by CD31 immunostaining.
RESULTS: The mean percentages of neovascularized corneal area in control mice and CCR2-deficient mice 2 weeks after denudation were 58.3% and 38.8% (P = 0.047), respectively.
CONCLUSIONS: Development of corneal neovascularization is inhibited in CCR2-deficient mice.

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Year:  2003        PMID: 12827053     DOI: 10.1097/00003226-200307000-00013

Source DB:  PubMed          Journal:  Cornea        ISSN: 0277-3740            Impact factor:   2.651


  13 in total

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9.  Deletion of the FHL2 gene attenuating neovascularization after corneal injury.

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10.  Nucleoside Reverse Transcriptase Inhibitors Suppress Laser-Induced Choroidal Neovascularization in Mice.

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