Literature DB >> 12813407

Loss of heterozygosity in patients with pseudoexfoliation syndrome.

Renata Zalewska1, Witold Pepinski, Danuta Smolenska-Janica, Zofia Mariak, Ewa Proniewska-Skretek, Malgorzata Skawronska, Jerzy Janica.   

Abstract

PURPOSE: The purpose of the study was to evaluate the possible occurrence of loss of heterozygosity (LOH) in the anterior capsule, lens nucleus, iris, and trabeculum samples taken from patients with pseudoexfoliation syndrome (PEX). Loss of heterozygosity in a microsatellite marker locus indicates that the neighboring gene may be inactivated. Previous attempts to find a gene defect that might be responsible for pseudoexfoliation glaucoma have been unsuccessful.
METHODS: Specimens of the anterior capsule, the lens nucleus, the iris, the trabeculum, and reference blood samples were collected from 19 PEX patients. Fluorescent multiplex PCR was used to amplify the microsatellite markers located on chromosomes 2, 4, 7, 12, 18, 19, and 21.
RESULTS: LOH was found in 58% of the iris specimens and 50% of the anterior capsule specimens collected from PEX patients. The highest incidence of LOH was observed at the marker D7S820.
CONCLUSIONS: It is possible, that genetic factors may be involved in the etiology and pathogenesis of PEX.

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Year:  2003        PMID: 12813407

Source DB:  PubMed          Journal:  Mol Vis        ISSN: 1090-0535            Impact factor:   2.367


  7 in total

1.  [Cataract patients with pseudoexfoliation (PEX) syndrome among a population with high prevalence of PEX. Clinical findings and morphological and surgical characteristics].

Authors:  A A Bialasiewicz; U Wali; R Shenoy; R Al-Saeidi
Journal:  Ophthalmologe       Date:  2005-12       Impact factor: 1.059

2.  [Patients with secondary open-angle glaucoma in pseudoexfoliation (PEX) syndrome among a population with high prevalence of PEX. Clinical findings and morphological and surgical characteristics].

Authors:  A A Bialasiewicz; U Wali; R Shenoy; R Al-Saeidi
Journal:  Ophthalmologe       Date:  2005-11       Impact factor: 1.059

3.  Lack of association between LOXL1 variants and primary open-angle glaucoma in three different populations.

Authors:  Yutao Liu; Silke Schmidt; Xuejun Qin; Jason Gibson; Kristen Hutchins; Cecile Santiago-Turla; Janey L Wiggs; Donald L Budenz; Stephen Akafo; Pratap Challa; Leon W Herndon; Michael A Hauser; R Rand Allingham
Journal:  Invest Ophthalmol Vis Sci       Date:  2008-04-17       Impact factor: 4.799

Review 4.  Emerging roles of oxidative stress in the pathogenesis of pseudoexfoliation syndrome (Review).

Authors:  Stylianos Mastronikolis; Marina Pagkalou; Panagiotis Plotas; Konstantinos Kagkelaris; Constantinos D Georgakopoulos
Journal:  Exp Ther Med       Date:  2022-07-28       Impact factor: 2.751

5.  TNF-alpha -308 G>A and -238 G>A polymorphisms are not major risk factors in Caucasian patients with exfoliation glaucoma.

Authors:  Georg Mossböck; Wilfried Renner; Yosuf El-Shabrawi; Christoph Faschinger; Otto Schmut; Andreas Wedrich; Christina Zimmermann; Martin Weger
Journal:  Mol Vis       Date:  2009-03-09       Impact factor: 2.367

6.  Lysyl oxidase-like 1 gene polymorphisms in Japanese patients with primary open angle glaucoma and exfoliation syndrome.

Authors:  Fumihiko Mabuchi; Yoichi Sakurada; Kenji Kashiwagi; Zentaro Yamagata; Hiroyuki Iijima; Shigeo Tsukahara
Journal:  Mol Vis       Date:  2008-07-14       Impact factor: 2.367

7.  Lysyl oxidase-like protein 1 (LOXL1) gene polymorphisms and exfoliation glaucoma in a Central European population.

Authors:  Georg Mossböck; Wilfried Renner; Christoph Faschinger; Otto Schmut; Andreas Wedrich; Martin Weger
Journal:  Mol Vis       Date:  2008-05-09       Impact factor: 2.367

  7 in total

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