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27 in total

1. Distinguishing modes of eukaryotic gradient sensing.

Authors: R Skupsky; W Losert; R J Nossal
Journal: Biophys J Date: 2005-08-05 Impact factor: 4.033

2. Phosphoinositide phosphatases and disease.

Authors: Philip W Majerus; John D York
Journal: J Lipid Res Date: 2008-11-11 Impact factor: 5.922

3. Inpp5f is a polyphosphoinositide phosphatase that regulates cardiac hypertrophic responsiveness.

Authors: Wenting Zhu; Chinmay M Trivedi; Diane Zhou; Lijun Yuan; Min Min Lu; Jonathan A Epstein
Journal: Circ Res Date: 2009-10-29 Impact factor: 17.367

4. Phosphoinositide 3-kinase p110δ promotes lumen formation through the enhancement of apico-basal polarity and basal membrane organization.

Authors: Juan Peng; Aline Awad; Sokhavuth Sar; Ola Hamze Komaiha; Romina Moyano; Amel Rayal; Didier Samuel; Annette Shewan; Bart Vanhaesebroeck; Keith Mostov; Ama Gassama-Diagne
Journal: Nat Commun Date: 2015-01-13 Impact factor: 14.919

Review 5. INPPL1 gene mutations in opsismodysplasia.

Authors: Anaïs Fradet; Jamie Fitzgerald
Journal: J Hum Genet Date: 2016-10-06 Impact factor: 3.172

6. A key role for the phosphorylation of Ser440 by the cyclic AMP-dependent protein kinase in regulating the activity of the Src homology 2 domain-containing Inositol 5'-phosphatase (SHIP1).

Authors: Jun Zhang; Kodi S Ravichandran; James C Garrison
Journal: J Biol Chem Date: 2010-09-01 Impact factor: 5.157

The termination of PI3K signalling by SHIP1 and SHIP2 inositol 5-phosphatases.

1. Distinguishing modes of eukaryotic gradient sensing.

2. Phosphoinositide phosphatases and disease.

3. Inpp5f is a polyphosphoinositide phosphatase that regulates cardiac hypertrophic responsiveness.

4. Phosphoinositide 3-kinase p110δ promotes lumen formation through the enhancement of apico-basal polarity and basal membrane organization.

Review 5. INPPL1 gene mutations in opsismodysplasia.

6. A key role for the phosphorylation of Ser440 by the cyclic AMP-dependent protein kinase in regulating the activity of the Src homology 2 domain-containing Inositol 5'-phosphatase (SHIP1).

7. SHIP2, a factor associated with diet-induced obesity and insulin sensitivity, attenuates FGF signaling in vivo.

8. Insulin hypersensitivity and resistance to streptozotocin-induced diabetes in mice lacking PTEN in adipose tissue.

9. Inositol phosphatase SHIP1 is a primary target of miR-155.

Review 10. PTEN function: how normal cells control it and tumour cells lose it.