Literature DB >> 12763292

Altered amino acid levels in sera of a mouse model for juvenile neuronal ceroid lipofuscinoses.

David A Pearce, Karin McCall, Robert A Mooney, Subrata Chattopadhyay, Timothy M Curran.   

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Year:  2003        PMID： 12763292     DOI： 10.1016/s0009-8981(03)00122-0

Source DB:  PubMed          Journal:  Clin Chim Acta        ISSN： 0009-8981            Impact factor:   3.786


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  4 in total

1.  Deletion of the Caenorhabditis elegans homologues of the CLN3 gene, involved in human juvenile neuronal ceroid lipofuscinosis, causes a mild progeric phenotype.

Authors:  G de Voer; P van der Bent; A J G Rodrigues; G-J B van Ommen; D J M Peters; P E M Taschner
Journal:  J Inherit Metab Dis       Date:  2005       Impact factor: 4.982

2.  Neuropeptide changes and neuroactive amino acids in CSF from humans and sheep with neuronal ceroid lipofuscinoses (NCLs, Batten disease).

Authors:  Graham W Kay; Marcel M Verbeek; Julie M Furlong; Michèl A A P Willemsen; David N Palmer
Journal:  Neurochem Int       Date:  2009-08-05       Impact factor: 3.921

3.  Nitric oxide signaling is disrupted in the yeast model for Batten disease.

Authors:  Nuno S Osório; Agostinho Carvalho; Agostinho J Almeida; Sérgio Padilla-Lopez; Cecília Leão; João Laranjinha; Paula Ludovico; David A Pearce; Fernando Rodrigues
Journal:  Mol Biol Cell       Date:  2007-05-02       Impact factor: 4.138

4.  A role in vacuolar arginine transport for yeast Btn1p and for human CLN3, the protein defective in Batten disease.

Authors:  Yoojin Kim; Denia Ramirez-Montealegre; David A Pearce
Journal:  Proc Natl Acad Sci U S A       Date:  2003-12-05       Impact factor: 11.205
  4 in total

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