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Literature DB >> 12747957

Endothelial hyperpermeability in vascular leakage.

Victor W M van Hinsbergh¹, Geerten P van Nieuw Amerongen.

Abstract

Mesh：

Year: 2002 PMID： 12747957 DOI： 10.1016/s1537-1891(03)00006-5

Source DB: PubMed Journal: Vascul Pharmacol ISSN： 1537-1891 Impact factor: 5.773

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10 in total

1. A 40-year-old man with recurrent fainting, hypotension, lower limb edema and oliguria with body weight gain and secondary erythrocytosis.

Authors: Giulia Berti de Marinis; Irene Bertozzi; Emanuele Allemand; Fabiana Tezza; Maria Luigia Randi; Agostino Naso; Fabrizio Fabris
Journal: Intern Emerg Med Date: 2011-09-25 Impact factor: 3.397

2. Role of vascular and lymphatic endothelial cells in hantavirus pulmonary syndrome suggests targeted therapeutic approaches.

Authors: Erich R Mackow; Elena E Gorbunova; Nadine A Dalrymple; Irina N Gavrilovskaya
Journal: Lymphat Res Biol Date: 2013-09-11 Impact factor: 2.589

Review 3. Inflammation in acute CNS injury: a focus on the role of substance P.

Authors: F Corrigan; R Vink; R J Turner
Journal: Br J Pharmacol Date: 2015-05-12 Impact factor: 8.739

4. Andes virus infection of lymphatic endothelial cells causes giant cell and enhanced permeability responses that are rapamycin and vascular endothelial growth factor C sensitive.

Authors: Irina N Gavrilovskaya; Elena E Gorbunova; Erich R Mackow
Journal: J Virol Date: 2012-06-13 Impact factor: 5.103

Endothelial hyperpermeability in vascular leakage.

1. A 40-year-old man with recurrent fainting, hypotension, lower limb edema and oliguria with body weight gain and secondary erythrocytosis.

2. Role of vascular and lymphatic endothelial cells in hantavirus pulmonary syndrome suggests targeted therapeutic approaches.

Review 3. Inflammation in acute CNS injury: a focus on the role of substance P.

4. Andes virus infection of lymphatic endothelial cells causes giant cell and enhanced permeability responses that are rapamycin and vascular endothelial growth factor C sensitive.

Review 5. Role of the cAMP-binding protein Epac in cardiovascular physiology and pathophysiology.

6. The Role of the Endothelium in HPS Pathogenesis and Potential Therapeutic Approaches.

7. Exposure to Concentrated Ambient Fine Particulate Matter Induces Vascular Endothelial Dysfunction via miR-21.

Review 8. The Therapeutic Potential of Targeting Substance P/NK-1R Interactions in Inflammatory CNS Disorders.

9. Endothelial cell senescence is associated with disrupted cell-cell junctions and increased monolayer permeability.

10. Phosphorylated cingulin localises GEF-H1 at tight junctions to protect vascular barriers in blood endothelial cells.