Literature DB >> 12745801

Adventitial myofibroblasts play no major role in neointima formation after angioplasty.

Michael Maeng1, Henrik Mertz, Søren Nielsen, Guillaume J J M van Eys, Klaus Rasmussen, Geert T Espersen.   

Abstract

OBJECTIVE: Myofibroblasts migrating from adventitia have been suggested to constitute a majority of neointimal cells after angioplasty. We sought to examine this hypothesis by use of smoothelin, which is a marker for the quiescent smooth muscle cell (SMC) phenotype while not expressed by myofibroblasts.
DESIGN: Balloon angioplasty was performed in left iliac arteries of 25 rabbits that were killed after 3-56 days. Arterial cross-sections were immunostained for alpha-actin (general marker), smoothelin (quiescent SMC phenotype), and Ki-67 (proliferative phenotype).
RESULTS: Adventitial cells became transiently actin-positive (myofibroblasts) but did not express smoothelin at any time point. In media, angioplasty induced transient proliferation and coinciding transient decrease in smoothelin expression. Neointimal cells, present 7 days after angioplasty, were initially proliferating and smoothelin-negative but changed to non-proliferating, smoothelin-positive cells after 56 days where 82 +/- 10% of cells stained positive for smoothelin. This phenotypic modulation of medial and intimal cells began in media and moved gradually towards the lumen.
CONCLUSION: At late follow-up, the majority of intimal cells are smoothelin-positive indicating that adventitial myofibroblasts play no major role for neointima formation.

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Year:  2003        PMID: 12745801     DOI: 10.1080/14017430310007018

Source DB:  PubMed          Journal:  Scand Cardiovasc J        ISSN: 1401-7431            Impact factor:   1.589


  3 in total

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Authors:  Sébastien Lepreux; Christelle Guyot; Fabrice Billet; Chantal Combe; Charles Balabaud; Paulette Bioulac-Sage; Alexis Desmoulière
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  3 in total

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