Literature DB >> 12722044

Effect of a 3-year therapy with the 3-hydroxy-3-methylglutaryl coenzyme a reductase-inhibitor fluvastatin on endothelial function and distensibility of large arteries in hypercholesterolemic renal transplant recipient.

Markus Kosch1, Michael Barenbrock, Barbara Suwelack, Roland M Schaefer, Karl-Heinz Rahn, Martin Hausberg.   

Abstract

BACKGROUND: In patients after renal transplantation functional arterial vessel wall properties are impaired. Whether 3-hydroxy-3-methylglutaryl coenzyme A (HMG-CoA) reductase inhibitors have a sustained effect on endothelial function and arterial distensibility in patients after renal transplantation is not clear. The authors studied the effects of a long-term therapy with fluvastatin on large artery distensibility and flow-mediated vasodilation (FMD) in hypercholesterolemic patients after renal transplantation in a prospective, blinded, and randomized trial.
METHODS: Twenty-six patients who had undergone renal transplantation were assigned randomly to either fluvastatin, 40 mg/d (n = 13) or placebo (n = 13) and underwent follow-up for 3 years. At baseline and after 6, 12, and 36 months of treatment, carotid and brachial artery distensibility, endothelium-dependent FMD, and nitroglycerine-induced vasodilation (NMD) of the brachial artery were measured by a echo-tracking device.
RESULTS: A significant decrease in total and low-density cholesterol was observed after 6, 12, and 36 months in patients treated with fluvastatin but not in the placebo group. FMD increased with fluvastatin from 4.6 +/- 2% to 12.4 +/- 2% after 12 months; this improvement was sustained with 13.4 +/- 3% after 36 months (P < 0.05). However, placebo did not alter FMD (P < 0.001 for trend difference between groups by analysis of covariance). Endothelium-independent NMD was similar in both groups at baseline and during therapy. Neither carotid nor brachial artery distensibility coefficients were altered by either treatment.
CONCLUSION: HMG-CoA reductase inhibitor therapy over 3 years results in a significant and sustained improvement of endothelial function in hypercholesterolemic patients after renal transplantation. However, this is not accompanied by a beneficial effect on impaired large artery distensibility even after long-term therapy with fluvastatin.

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Year:  2003        PMID: 12722044     DOI: 10.1016/s0272-6386(03)00207-5

Source DB:  PubMed          Journal:  Am J Kidney Dis        ISSN: 0272-6386            Impact factor:   8.860


  3 in total

1.  Beneficial vasoactive endothelial effects of fluvastatin: focus on prostacyclin and nitric oxide.

Authors:  Cristine Skogastierna; Leonid Luksha; Karolina Kublickiene; Erik Eliasson; Anders Rane; Lena Ekström
Journal:  Heart Vessels       Date:  2011-01-07       Impact factor: 2.037

2.  Progression of kidney disease in moderately hypercholesterolemic, hypertensive patients randomized to pravastatin versus usual care: a report from the Antihypertensive and Lipid-Lowering Treatment to Prevent Heart Attack Trial (ALLHAT).

Authors:  Mahboob Rahman; Charles Baimbridge; Barry R Davis; Joshua Barzilay; Jan N Basile; Mario A Henriquez; Anne Huml; Nelson Kopyt; Gail T Louis; Sara L Pressel; Clive Rosendorff; Sithiporn Sastrasinh; Carol Stanford
Journal:  Am J Kidney Dis       Date:  2008-08-03       Impact factor: 8.860

Review 3.  HMG CoA reductase inhibitors (statins) for kidney transplant recipients.

Authors:  Suetonia C Palmer; Sankar D Navaneethan; Jonathan C Craig; Vlado Perkovic; David W Johnson; Sagar U Nigwekar; Jorgen Hegbrant; Giovanni Fm Strippoli
Journal:  Cochrane Database Syst Rev       Date:  2014-01-28
  3 in total

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