Literature DB >> 12673711

Analysis of the Na,K-ATPase alpha- and beta-subunit expression profiles of bladder cancer using tissue microarrays.

Cromwell Espineda1, David B Seligson, William James Ball, JianYu Rao, Aarno Palotie, Steve Horvath, Yunda Huang, Tao Shi, Ayyappan K Rajasekaran.   

Abstract

BACKGROUND: The purpose of this study was to determine the clinical significance of Na,K-ATPase alpha- and beta-subunit expression in a histopathologically well-characterized group of patients representing a wide spectrum of tumor grades and disease stages with transitional cell carcinomas (TCC).
METHODS: Na,K-ATPase alpha- and beta-subunit protein expression patterns were analyzed using immunohistochemistry on urothelial cancer tissue microarrays (TMA) of 146 patients diagnosed with urothelial carcinoma. For each subunit, the maximum staining intensity and the percentage of positive cells staining at the maximal intensity were analyzed.
RESULTS: Compared with the benign fields, the mean protein expression for both Na,K-ATPase alpha- and beta-subunits were found to be decreased overall in in situ and invasive tumors, as well as in tumor-adjacent dysplastic fields. When Na,K-ATPase alpha- and beta-subunit expression levels were dichotomized into distinct groups, they were both found to be significant predictors of recurrence risk in multivariate logistic regression analysis (P = 0.0062, odds ratio [OR] = 2.6 and P = 0.013, OR = 0.43, for Na,K-ATPase alpha- and beta-subunits, respectively). The authors also found that patients with high alpha- and low beta-subunit expression had a high risk for early recurrence, whereas patients with a low alpha- and high beta-subunit expression had a significantly longer median recurrence-free time (17 months and 125 months, respectively, log rank statistics P = 0.0005).
CONCLUSIONS: The results suggested that Na,K-ATPase alpha- and beta-subunit expression levels may be useful predictors of clinical outcomes such as recurrence-free time of bladder cancer patients. Copyright 2003 American Cancer Society.

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Year:  2003        PMID: 12673711     DOI: 10.1002/cncr.11267

Source DB:  PubMed          Journal:  Cancer        ISSN: 0008-543X            Impact factor:   6.860


  32 in total

1.  Na,K-ATPase subunits as markers for epithelial-mesenchymal transition in cancer and fibrosis.

Authors:  Sigrid A Rajasekaran; Thu P Huynh; Daniel G Wolle; Cromwell E Espineda; Landon J Inge; Anna Skay; Charles Lassman; Susanne B Nicholas; Jeffrey F Harper; Anna E Reeves; Mansoor M Ahmed; James M Leatherman; James M Mullin; Ayyappan K Rajasekaran
Journal:  Mol Cancer Ther       Date:  2010-05-25       Impact factor: 6.261

2.  Novel role for Na,K-ATPase in phosphatidylinositol 3-kinase signaling and suppression of cell motility.

Authors:  Sonali P Barwe; Gopalakrishnapillai Anilkumar; Sun Y Moon; Yi Zheng; Julian P Whitelegge; Sigrid A Rajasekaran; Ayyappan K Rajasekaran
Journal:  Mol Biol Cell       Date:  2004-12-22       Impact factor: 4.138

3.  Na,K-ATPase is a target of cigarette smoke and reduced expression predicts poor patient outcome of smokers with lung cancer.

Authors:  Thu P Huynh; Vei Mah; Valerie B Sampson; David Chia; Michael C Fishbein; Steve Horvath; Mohammad Alavi; Debbie C Wu; Jeffrey Harper; Ted Sarafian; Steven M Dubinett; Sigrid A Langhans; Lee Goodglick; Ayyappan K Rajasekaran
Journal:  Am J Physiol Lung Cell Mol Physiol       Date:  2012-02-17       Impact factor: 5.464

4.  Na/K-ATPase mimetic pNaKtide peptide inhibits the growth of human cancer cells.

Authors:  Zhichuan Li; Zhongbing Zhang; Joe X Xie; Xin Li; Jiang Tian; Ting Cai; Hongjuan Cui; Hanfei Ding; Joseph I Shapiro; Zijian Xie
Journal:  J Biol Chem       Date:  2011-07-22       Impact factor: 5.157

5.  Cellular location and expression of Na+, K+ -ATPase α subunits affect the anti-proliferative activity of oleandrin.

Authors:  Peiying Yang; Carrie Cartwright; Ekem Efuet; Stanley R Hamilton; Ignacio Ivan Wistuba; David Menter; Crandell Addington; Imad Shureiqi; Robert A Newman
Journal:  Mol Carcinog       Date:  2012-10-16       Impact factor: 4.784

6.  Regulation of Na,K-ATPase β1-subunit in TGF-β2-mediated epithelial-to-mesenchymal transition in human retinal pigmented epithelial cells.

Authors:  Sridevi Mony; Seung Joon Lee; Jeffrey F Harper; Sonali P Barwe; Sigrid A Langhans
Journal:  Exp Eye Res       Date:  2013-06-28       Impact factor: 3.467

7.  Evidence for a potential tumor suppressor role for the Na,K-ATPase beta1-subunit.

Authors:  L J Inge; S A Rajasekaran; K Yoshimoto; P S Mischel; W McBride; E Landaw; A K Rajasekaran
Journal:  Histol Histopathol       Date:  2008-04       Impact factor: 2.303

8.  "The Lower Threshold" phenomenon in tumor cells toward endogenous digitalis-like compounds: Responsible for tumorigenesis?

Authors:  Heidrun Weidemann
Journal:  J Carcinog       Date:  2012-02-17

9.  Tumor Tissue Oxidative Stress Changes and Na, K-ATPase Evaluation in Head and Neck Squamous Cell Carcinoma.

Authors:  Marina Marques Toledo; Bruno De Souza Gonçalves; Natalie Mounteer Colodette; Aline Lauda Freitas Chaves; Luciana Vieira Muniz; Rosy Iara Maciel De A Ribeiro; Hélio Batista Dos Santos; Vanessa F Cortes; Joao Marcos Arantes Soares; Hérica De Lima Santos; Leandro A Barbosa
Journal:  J Membr Biol       Date:  2021-06-08       Impact factor: 1.843

10.  Trial watch: Cardiac glycosides and cancer therapy.

Authors:  Laurie Menger; Erika Vacchelli; Oliver Kepp; Alexander Eggermont; Eric Tartour; Laurence Zitvogel; Guido Kroemer; Lorenzo Galluzzi
Journal:  Oncoimmunology       Date:  2013-02-01       Impact factor: 8.110

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