Literature DB >> 12606820

Modulation of trophoblast cell death by oxygen and EGF.

Jonathan Perkins1, Justin St John, Asif Ahmed.   

Abstract

BACKGROUND: Preeclampsia, a maternal hypertensive disease, is characterized by shallow invasion of the maternal spiral arterioles resulting in hypoxia/reperfusion type insult; however, the molecular mechanism is unknown. The aim of this study was to determine the mechanism of altered oxygen tension or inhibition of phosphatidyl-inositol-3-kinase (PI3K) on trophoblast survival and to investigate the effect of epidermal growth factor (EGF) on maintaining cellular integrity. MATERIALS AND
METHOD: We have used flow cytometry, immunoblotting, and fluoroimmunocytochemistry to study apoptosis in a characterized, spontaneously transformed first trimester extravillous-like trophoblast cell line that exhibits many characteristics of in vivo trophoblast.
RESULTS: Time-dependent exposure of first trimester extravillous-like trophoblast to all oxygen tensions tested promoted dissipation of the mitochondrial membrane potential (psi(m)) and resulted in a significant increase in celldeath by 48 hr as determined by dual staining flow cytometry. Western blot analysis revealed expression ofcleaved caspase-3 and caspase-9 increased with time with hypoxia and hyperoxia promoting the greatest elevation indicating that longer duration of exposure to a change inoxygen tension causes increased apoptosis via a mitochondrial-mediated pathway. Disruption of the anti-apoptotic PI3K pathway by LY294002 (40 microM), its specific inhibitor, caused further significant dissipation of the psi(m) (p< 0.01) and cleavage of caspase-3. EGF was able to maintain the psi(m) and to prevent cleavage of caspase-3 even in the presence of LY294002, indicating that its survival effects were independent of the PI3K pathway.
CONCLUSIONS: These results suggest that inhibition of the PI3K/Akt pathway can sensitize first-trimester trophoblast-like cells into oxygen-induced cell death and that EGF exerts its anti-apoptotic effect independently of PI3K/Akt.

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Year:  2002        PMID: 12606820      PMCID: PMC2039959     

Source DB:  PubMed          Journal:  Mol Med        ISSN: 1076-1551            Impact factor:   6.354


  5 in total

1.  AKT controls human first trimester trophoblast cell sensitivity to FAS-mediated apoptosis by regulating XIAP expression.

Authors:  Shawn L Straszewski-Chavez; Vikki M Abrahams; Paula B Aldo; Roberto Romero; Gil Mor
Journal:  Biol Reprod       Date:  2009-09-02       Impact factor: 4.285

2.  STOX1 overexpression in choriocarcinoma cells mimics transcriptional alterations observed in preeclamptic placentas.

Authors:  Virginie Rigourd; Caroline Chauvet; Sonia T Chelbi; Régis Rebourcet; Françoise Mondon; Franck Letourneur; Thérèse-Marie Mignot; Sandrine Barbaux; Daniel Vaiman
Journal:  PLoS One       Date:  2008-12-11       Impact factor: 3.240

3.  The role of VEGF-A165b in trophoblast survival.

Authors:  Victoria L Bills; Maryam Hamdollah-Zadeh; Peter W Soothill; Steven J Harper; David O Bates
Journal:  BMC Pregnancy Childbirth       Date:  2014-08-15       Impact factor: 3.007

4.  Requirement of Leukemia Inhibitory Factor or Epidermal Growth Factor for Pre-Implantation Embryogenesis via JAK/STAT3 Signaling Pathways.

Authors:  En-Hui Cheng; Jer-Yuh Liu; Tsung-Hsein Lee; Chun-Chia Huang; Chung-I Chen; Lii-Sheng Huang; Maw-Sheng Lee
Journal:  PLoS One       Date:  2016-04-20       Impact factor: 3.240

5.  Age and Sex-Related Changes in Human First-Trimester Placenta Transcriptome and Insights into Adaptative Responses to Increased Oxygen.

Authors:  Fulin Liu; Christelle Simasotchi; Françoise Vibert; Wencan Zhu; Sophie Gil; Séverine A Degrelle; Thierry Fournier
Journal:  Int J Mol Sci       Date:  2021-03-12       Impact factor: 5.923

  5 in total

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