Literature DB >> 12593628

Neuroprotection by the stable nitroxide 3-carbamoyl-proxyl during reperfusion in a rat model of transient focal ischemia.

Guohan Hu1, Bruce G Lyeth, Xueren Zhao, James B Mitchell, Joe C Watson.   

Abstract

OBJECT: Nitroxides mimic superoxide dismutase (SOD) biochemically and may prevent free radical oxidative injury in settings in which endogenous SOD is overwhelmed. The authors have previously shown the efficacy of a nitroxide, Tempol, in reducing stroke infarct size. Of the nitroxides, 3-carbamoyl-proxyl (3-CP) is especially promising for clinical use, because it does not cause hypotension in animals. Its efficacy in brain ischemia, however, is untested. The goal of this study was to ascertain whether 3-CP would reduce brain damage in a rat ischemia-reperfusion model.
METHODS: The authors performed a blinded, dose-response study of the effect of different amounts of 3-CP (1, 10, and 100 mg/kg) on infarct size at 24 hours after focal ischemia and reperfusion. The 3-CP was given intravenously during reperfusion, which followed 1 hour of reversible ischemia induced by a thread placed intraluminally in the middle cerebral artery of rats. Brain infarcts, measured with 2,3,5-triphenyltetrazolium chloride staining in six 3-CP groups, were compared with those measured in controls (animals given an equal volume of saline). Edema-corrected infarct sizes (mean +/- standard deviation) were as follows: 146 +/- 64 mm3 in controls; 107 +/- 18 mm3 in rats given 1 mg/kg 3-CP; 40 +/- 20 mm3 in those given 10 mg/kg 3-CP; and 44 +/- 17 mm3 in those given 100 mg/kg 3-CP. A statistically significant reduction in infarct size was achieved in the 10- and 100-mg/kg 3-CP-treated groups (p < 0.01). A reduction in infarct size was also seen in the 1 mg/kg 3-CP-treated group, but this did not reach statistical significance. The authors observed no effects of 3-CP on blood pressure or brain temperature.
CONCLUSIONS: Given at reperfusion, 3-CP significantly decreases brain infarct size at doses of 10 and 100 mg/kg without causing hypotension. The authors found that 3-CP is well suited for further laboratory and clinical use in brain ischemia and reperfusion.

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Year:  2003        PMID: 12593628     DOI: 10.3171/jns.2003.98.2.0393

Source DB:  PubMed          Journal:  J Neurosurg        ISSN: 0022-3085            Impact factor:   5.115


  4 in total

Review 1.  Effects of tempol and redox-cycling nitroxides in models of oxidative stress.

Authors:  Christopher S Wilcox
Journal:  Pharmacol Ther       Date:  2010-02-11       Impact factor: 12.310

2.  Ginsenoside Rg1 attenuates cerebral ischemia-reperfusion injury due to inhibition of NOX2-mediated calcium homeostasis dysregulation in mice.

Authors:  Yuli Han; Xuewang Li; Liu Yang; Duoduo Zhang; Lan Li; Xianan Dong; Yan Li; Sen Qun; Weizu Li
Journal:  J Ginseng Res       Date:  2021-08-10       Impact factor: 5.735

3.  Tempol-mediated activation of latent iron regulatory protein activity prevents symptoms of neurodegenerative disease in IRP2 knockout mice.

Authors:  Manik C Ghosh; Wing-Hang Tong; Deliang Zhang; Hayden Ollivierre-Wilson; Anamika Singh; Murali C Krishna; James B Mitchell; Tracey A Rouault
Journal:  Proc Natl Acad Sci U S A       Date:  2008-08-06       Impact factor: 11.205

4.  In vivo redox metabolic imaging of mitochondria assesses disease progression in non-alcoholic steatohepatitis.

Authors:  Ryosuke Nakata; Fuminori Hyodo; Masaharu Murata; Hinako Eto; Tomoko Nakaji; Takahito Kawano; Sayoko Narahara; Keiji Yasukawa; Tomohiko Akahoshi; Morimasa Tomikawa; Makoto Hashizume
Journal:  Sci Rep       Date:  2017-12-07       Impact factor: 4.379

  4 in total

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