BACKGROUND: The effect on joint stability of repair of type II superior labrum and biceps anchor lesions is unknown. HYPOTHESIS: Increased translations of the glenohumeral joint after a simulated type II lesion will be reduced after the lesion is repaired. STUDY DESIGN: Controlled laboratory study. METHODS: A robotic/universal force-moment testing system was used to simulate load-and-shift and apprehension tests on eight cadaveric shoulders to determine joint kinematics of the shoulder after venting, creation of a type II lesion, and repair of the lesion. RESULTS: At 30 degrees of abduction, anterior translation of the vented joint in response to an anterior load was 18.7 +/- 8.5 mm and was significantly increased to 26.2 +/- 6.5 mm after simulation of a type II lesion. Repair did not restore anterior translation (23.9 +/- 8.6 mm) to that of the vented joint. The inferior translation that also occurred during application of an anterior load was 3.8 +/- 4.0 mm in the vented joint and increased significantly to 8.5 +/- 5.4 mm with a simulated type II lesion. After repair, the inferior translation decreased significantly to 6.7 +/- 5.3 mm. CONCLUSIONS: Repair of a type II lesion only partially restored glenohumeral translations to that of the vented joint. CLINICAL RELEVANCE: Surgical techniques including improved repair of passive stabilizers injured in the type II lesion should be considered. Copyright 2003 American Orthopaedic Society for Sports Medicine
BACKGROUND: The effect on joint stability of repair of type II superior labrum and biceps anchor lesions is unknown. HYPOTHESIS: Increased translations of the glenohumeral joint after a simulated type II lesion will be reduced after the lesion is repaired. STUDY DESIGN: Controlled laboratory study. METHODS: A robotic/universal force-moment testing system was used to simulate load-and-shift and apprehension tests on eight cadaveric shoulders to determine joint kinematics of the shoulder after venting, creation of a type II lesion, and repair of the lesion. RESULTS: At 30 degrees of abduction, anterior translation of the vented joint in response to an anterior load was 18.7 +/- 8.5 mm and was significantly increased to 26.2 +/- 6.5 mm after simulation of a type II lesion. Repair did not restore anterior translation (23.9 +/- 8.6 mm) to that of the vented joint. The inferior translation that also occurred during application of an anterior load was 3.8 +/- 4.0 mm in the vented joint and increased significantly to 8.5 +/- 5.4 mm with a simulated type II lesion. After repair, the inferior translation decreased significantly to 6.7 +/- 5.3 mm. CONCLUSIONS: Repair of a type II lesion only partially restored glenohumeral translations to that of the vented joint. CLINICAL RELEVANCE: Surgical techniques including improved repair of passive stabilizers injured in the type II lesion should be considered. Copyright 2003 American Orthopaedic Society for Sports Medicine
Authors: T Patzer; P Habermeyer; C Hurschler; E Bobrowitsch; J R Paletta; S Fuchs-Winkelmann; M D Schofer Journal: Knee Surg Sports Traumatol Arthrosc Date: 2011-02-22 Impact factor: 4.342
Authors: Susan M Moore; Benjamin Ellis; Jeffrey A Weiss; Patrick J McMahon; Richard E Debski Journal: Ann Biomed Eng Date: 2009-11-13 Impact factor: 3.934