Literature DB >> 125309

The production of cardiac hypertrophy by tri-iodothyroacetic acid.

C Symons, E G Olsen, C M Hawkey.   

Abstract

Severe cardiac hypertrophy has been produced experimentally in rats by long-term, low-dose treatment with tri-iodothyroacetic acid. The dose used was insufficient to cause any apparent systemic or metabolic effect. It is suggested that similar iodinated substances in the blood in man, resulting from normal or abnormal thyroid hormone catabolism, may be causally related to some forms of cardiomyopathy.

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Year:  1975        PMID: 125309     DOI: 10.1677/joe.0.0650341

Source DB:  PubMed          Journal:  J Endocrinol        ISSN: 0022-0795            Impact factor:   4.286


  5 in total

1.  The effects of plasma from patients with Graves' disease on foetal mouse hearts in organ culture.

Authors:  A W Nathan; D B Longmore; C W Havard; P Dandona
Journal:  Br J Exp Pathol       Date:  1983-10

2.  Thyroid heart disease.

Authors:  C Symons
Journal:  Br Heart J       Date:  1979-03

3.  Response to thyrotrophin-releasing hormone in atrial dysrhythmias.

Authors:  C Symons; A Myers; D Kingstone; M Boss
Journal:  Postgrad Med J       Date:  1978-10       Impact factor: 2.401

4.  3-Iodothyroacetic acid lacks thermoregulatory and cardiovascular effects in vivo.

Authors:  Carolin S Hoefig; Simon F Jacobi; Amy Warner; Lisbeth Harder; Nancy Schanze; Björn Vennström; Jens Mittag
Journal:  Br J Pharmacol       Date:  2015-06-09       Impact factor: 8.739

Review 5.  Myocardial diseases of animals.

Authors:  J F Van Vleet; V J Ferrans
Journal:  Am J Pathol       Date:  1986-07       Impact factor: 4.307

  5 in total

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