Literature DB >> 12505216

Enhanced extracellular matrix accumulation in restenosis of coronary arteries after stent deployment.

Ick-Mo Chung1, Herman K Gold, Stephen M Schwartz, Yuji Ikari, Michael A Reidy, Thomas N Wight.   

Abstract

OBJECTIVES: The goal of this study was to evaluate the cellular and extracellular composition of human coronary arterial in-stent restenosis after various periods of time following stent deployment.
BACKGROUND: Neointimal in-growth rather than stent recoil is thought to be important for coronary arterial in-stent restenosis. There is only limited data on the cellular and extracellular composition changes with time after stent deployment.
METHODS: We analyzed 29 coronary arterial in-stent restenotic tissue samples (14 left anterior descending coronary artery, 10 right coronary artery, and 5 left circumflex artery) retrieved by using directional coronary atherectomy from 25 patients at 0.5 to 23 (mean, 5.7) months after deployment of Palmaz-Schatz stents employing histochemical and immunocytochemical techniques.
RESULTS: Cell proliferation was low (0% to 4%). Myxoid tissue containing extracellular matrix (ECM) enriched with proteoglycans was found in 69% of cases and decreased over time after stenting. Cell-depleted areas were found in 57% of cases and increased with time after stenting. Versican, biglycan, perlecan, and hyaluronan were present with varying individual distributions in all samples. Positive transforming growth factor-beta1 staining was found in 80% of cases. Immunostaining with alpha-smooth muscle actin identified the majority of cells as smooth muscle cells with occasional macrophages present (< or =12 cells per section).
CONCLUSIONS: These data suggest that enhanced ECM accumulation rather than cell proliferation contribute to later stages of in-stent restenosis. Balloon angioplasty of in-stent restenosis may, therefore, fail due to ECM changes during: 1) additional stent expansion, 2) tissue extrusion out of the stent, or 3) tissue compression.

Entities:  

Mesh:

Substances:

Year:  2002        PMID: 12505216     DOI: 10.1016/s0735-1097(02)02598-6

Source DB:  PubMed          Journal:  J Am Coll Cardiol        ISSN: 0735-1097            Impact factor:   24.094


  39 in total

1.  Impact of intima re-intrusion and expansion within 100 minutes on late lumen loss in percutaneous coronary intervention for diffuse in-stent restenosis.

Authors:  R Iijima; Y Ikari; T Tsunoda; M Nakamura; K Hara; T Yamaguchi
Journal:  Heart       Date:  2004-09       Impact factor: 5.994

2.  TiO2-Based Nanotopographical Cues Attenuate the Restenotic Phenotype in Primary Human Vascular Endothelial and Smooth Muscle Cells.

Authors:  Yiqi Cao; Tejal A Desai
Journal:  ACS Biomater Sci Eng       Date:  2020-01-17

Review 3.  Current understanding of coronary in-stent restenosis. Pathophysiology, clinical presentation, diagnostic work-up, and management.

Authors:  T M Schiele
Journal:  Z Kardiol       Date:  2005-11

4.  Intracoronary photodynamic therapy reduces neointimal growth without suppressing re-endothelialisation in a porcine model.

Authors:  R Waksman; I M Leitch; J Roessler; H Yazdi; R Seabron; F Tio; R W Scott; R I Grove; S Rychnovsky; B Robinson; R Pakala; E Cheneau
Journal:  Heart       Date:  2006-01-06       Impact factor: 5.994

5.  Hemocoagulase atrox reduces vascular modeling in rabbit carotid artery adventitia.

Authors:  Sheng-Yun Wan; Yuan-Cheng Hu; Yan-Qing Zhan; Dan-Dan Qin; Yang Ding
Journal:  Int J Clin Exp Pathol       Date:  2013-10-15

6.  Chronic thrombus occlusion after bare metal stenting in a patient with acute coronary syndrome visualized by optical coherence tomography.

Authors:  Tomonori Itoh; Tetsuya Fusazaki; Hiroki Oikawa; Takumi Kimura; Iwao Goto; Hiroki Matsui; Shoma Sugawara; Motoyuki Nakamura
Journal:  J Cardiol Cases       Date:  2010-08-26

7.  Sirolimus blocks the accumulation of hyaluronan (HA) by arterial smooth muscle cells and reduces monocyte adhesion to the ECM.

Authors:  Yann Gouëffic; Susan Potter-Perigo; Christina K Chan; Pamela Y Johnson; Kathleen Braun; Steven P Evanko; Thomas N Wight
Journal:  Atherosclerosis       Date:  2006-12-14       Impact factor: 5.162

8.  MT1-MMP evaluation in neointimal hyperplasia in the late follow-up after prosthesis implantation.

Authors:  Marta Bruczko; Tomasz Gogiel; Małgorzata Wolańska; Radosław Kowalewski; Krzysztof Sobolewski; Lech Romanowicz
Journal:  Int J Exp Pathol       Date:  2019-05-06       Impact factor: 1.925

9.  Athsq1 is an atherosclerosis modifier locus with dramatic effects on lesion area and prominent accumulation of versican.

Authors:  Sara Bretschger Seidelmann; Chaoling Kuo; Nick Pleskac; Jennifer Molina; Scott Sayers; Rong Li; Jing Zhou; Pamela Johnson; Kathleen Braun; Christina Chan; Daniel Teupser; Jan L Breslow; Thomas N Wight; Alan R Tall; Carrie L Welch
Journal:  Arterioscler Thromb Vasc Biol       Date:  2008-09-25       Impact factor: 8.311

10.  Blockade of TGF-β by catheter-based local intravascular gene delivery does not alter the in-stent neointimal response, but enhances inflammation in pig coronary arteries.

Authors:  Ick-Mo Chung; Junwoo Kim; Youngmi K Pak; Yangsoo Jang; Woo-Ick Yang; Innoc Han; Seung-Jung Park; Seong-Wook Park; Jooryung Huh; Thomas N Wight; Hikaru Ueno
Journal:  Int J Cardiol       Date:  2010-01-06       Impact factor: 4.164

View more

北京卡尤迪生物科技股份有限公司 © 2022-2023.