Warning: Undefined array key "mm" in /www/wwwroot/www.ai-bt.com/si.php on line 10 Deprecated: trim(): Passing null to parameter #1 ($string) of type string is deprecated in /www/wwwroot/www.ai-bt.com/si.php on line 10 Bax, along with lipid conspirators, allows cytochrome c to escape mitochondria.

Literature DB >> 12453403

Bax, along with lipid conspirators, allows cytochrome c to escape mitochondria.

J Marie Hardwick¹, Brian M Polster.

Abstract

Entities: Chemical Gene

Mesh：

Substances：

Year: 2002 PMID： 12453403 DOI： 10.1016/s1097-2765(02)00751-7

Source DB: PubMed Journal: Mol Cell ISSN： 1097-2765 Impact factor: 17.970

Keyword Cloud
Cited

× No keyword cloud information.

11 in total

1. Modulation of synaptic transmission by the BCL-2 family protein BCL-xL.

Authors: Elizabeth A Jonas; Daniel Hoit; John A Hickman; Teresa A Brandt; Brian M Polster; Yihru Fannjiang; Erin McCarthy; Marlena K Montanez; J Marie Hardwick; Leonard K Kaczmarek
Journal: J Neurosci Date: 2003-09-10 Impact factor: 6.167

2. Lack of Bax prevents influenza A virus-induced apoptosis and causes diminished viral replication.

Authors: Jeffrey E McLean; Emmanuel Datan; Demetrius Matassov; Zahra F Zakeri
Journal: J Virol Date: 2009-06-03 Impact factor: 5.103

3. Mitochondrial outer membrane proteins assist Bid in Bax-mediated lipidic pore formation.

Authors: Blanca Schafer; Joel Quispe; Vineet Choudhary; Jerry E Chipuk; Teddy G Ajero; Han Du; Roger Schneiter; Tomomi Kuwana
Journal: Mol Biol Cell Date: 2009-02-25 Impact factor: 4.138

Review 4. Mitochondrial regulation of cell death.

Authors: Stephen W G Tait; Douglas R Green
Journal: Cold Spring Harb Perspect Biol Date: 2013-09-01 Impact factor: 10.005

5. Drp1 is dispensable for apoptotic cytochrome c release in primed MCF10A and fibroblast cells but affects Bcl-2 antagonist-induced respiratory changes.

Authors: P Clerc; S X Ge; H Hwang; J Waddell; B A Roelofs; M Karbowski; H Sesaki; B M Polster
Journal: Br J Pharmacol Date: 2014-04 Impact factor: 8.739

10. Application of flow cytometry to determine differential redistribution of cytochrome c and Smac/DIABLO from mitochondria during cell death signaling.

Authors: Heling Ng; Danielle J Smith; Phillip Nagley
Journal: PLoS One Date: 2012-07-27 Impact factor: 3.240

Bax, along with lipid conspirators, allows cytochrome c to escape mitochondria.

1. Modulation of synaptic transmission by the BCL-2 family protein BCL-xL.

2. Lack of Bax prevents influenza A virus-induced apoptosis and causes diminished viral replication.

3. Mitochondrial outer membrane proteins assist Bid in Bax-mediated lipidic pore formation.

Review 4. Mitochondrial regulation of cell death.

5. Drp1 is dispensable for apoptotic cytochrome c release in primed MCF10A and fibroblast cells but affects Bcl-2 antagonist-induced respiratory changes.

Review 6. Mitochondrial death pathways in yeast and mammalian cells.

7. Inhibition of Bax-induced cytochrome c release from neural cell and brain mitochondria by dibucaine and propranolol.

Review 8. Mitochondria and cell death: outer membrane permeabilization and beyond.

9. Fatty acids enhance membrane permeabilization by pro-apoptotic Bax.

10. Application of flow cytometry to determine differential redistribution of cytochrome c and Smac/DIABLO from mitochondria during cell death signaling.