Literature DB >> 12444281

Strong Stromal Hyaluronan Expression Is Associated with PSA Recurrence in Local Prostate Cancer.

S Aaltomaa1, P Lipponen, R Tammi, M Tammi, J Viitanen, J-P Kankkunen, V-M Kosma.   

Abstract

OBJECTIVE: HA (hyaluronan) is involved in cell migration, differentiation and cell proliferation, which all are essential to tumour growth. In addition, the cell surface receptor of HA, CD44, is important in cancer cell adhesion, cell migration and tumour neovascularisation. We studied the expression of HA and CD44 and their relationship with other prognostic factors and prostate-specific antigen (PSA) recurrence in local prostate cancer (PC).
MATERIALS AND METHODS: 77 PC patients treated with radical prostatectomy were followed-up for a mean of 4 years. HA was detected by using a HA specific probe and CD44 expression was analysed by conventional immunohistochemistry.
RESULTS: All specimens expressed HA in tumour stroma and 78% (60/77) of the tumours showed strong stromal expression of HA. The fraction of positively stained specimens for CD44 was 66% (51/77). The strong stromal HA expression was related to perineural infiltration (p = 0.001) and capsule invasion (p = 0.05). No correlation was demonstrated between the stromal HA expression and CD44 expression, preoperative PSA, clinical or pathological T classification, pN status, Gleason grade, seminal vesicle invasion or surgical margin invasion. Reduced CD44 expression was related only to preoperative PSA level (p = 0.008). The PSA recurrence was predicted by strong stromal HA expression, pT classification, seminal vesicle invasion, capsule invasion and surgical margin invasion (p <or= 0.02 for all). pT classification and seminal vesicle infiltration predicted PSA recurrence free survival (RFS). In the multivariate analysis only the seminal vesicle infiltration was an independent predictor of PSA RFS (p = 0.009).
CONCLUSION: The strong stromal HA expression was related to PSA recurrence, perineural infiltration and seminal vesicle invasion. Those are the factors related to unfavourable prognosis, but the follow-up period of this series is too short to make definitive prognostic conclusions. Copyright 2002 S. Karger AG, Basel

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Year:  2002        PMID: 12444281     DOI: 10.1159/000066123

Source DB:  PubMed          Journal:  Urol Int        ISSN: 0042-1138            Impact factor:   2.089


  29 in total

1.  Antitumor activity of hyaluronic acid synthesis inhibitor 4-methylumbelliferone in prostate cancer cells.

Authors:  Vinata B Lokeshwar; Luis E Lopez; Daniel Munoz; Andrew Chi; Samir P Shirodkar; Soum D Lokeshwar; Diogo O Escudero; Neetika Dhir; Norman Altman
Journal:  Cancer Res       Date:  2010-03-23       Impact factor: 12.701

2.  Concurrent expression of hyaluronan biosynthetic and processing enzymes promotes growth and vascularization of prostate tumors in mice.

Authors:  Melanie A Simpson
Journal:  Am J Pathol       Date:  2006-07       Impact factor: 4.307

3.  Stromal hyaluronan interaction with epithelial CD44 variants promotes prostate cancer invasiveness by augmenting expression and function of hepatocyte growth factor and androgen receptor.

Authors:  Shibnath Ghatak; Vincent C Hascall; Roger R Markwald; Suniti Misra
Journal:  J Biol Chem       Date:  2010-03-03       Impact factor: 5.157

4.  Hyaluronidase Hyal1 Increases Tumor Cell Proliferation and Motility through Accelerated Vesicle Trafficking.

Authors:  Caitlin O McAtee; Abigail R Berkebile; Christian G Elowsky; Teresa Fangman; Joseph J Barycki; James K Wahl; Oleh Khalimonchuk; Naava Naslavsky; Steve Caplan; Melanie A Simpson
Journal:  J Biol Chem       Date:  2015-04-08       Impact factor: 5.157

5.  A PSP94-derived peptide PCK3145 inhibits MMP-9 secretion and triggers CD44 cell surface shedding: implication in tumor metastasis.

Authors:  Borhane Annabi; Mounia Bouzeghrane; Jean-Christophe Currie; Robert Hawkins; Hélène Dulude; Luc Daigneault; Marcia Ruiz; Jan Wisniewski; Seema Garde; Shafaat A Rabbani; Chandra Panchal; Jinzi J Wu; Richard Béliveau
Journal:  Clin Exp Metastasis       Date:  2005       Impact factor: 5.150

6.  Prostate cancer increases hyaluronan in surrounding nonmalignant stroma, and this response is associated with tumor growth and an unfavorable outcome.

Authors:  Andreas Josefsson; Hani Adamo; Peter Hammarsten; Torvald Granfors; Pär Stattin; Lars Egevad; Anna Engström Laurent; Pernilla Wikström; Anders Bergh
Journal:  Am J Pathol       Date:  2011-08-18       Impact factor: 4.307

Review 7.  Hyaluronan: genetic insights into the complex biology of a simple polysaccharide.

Authors:  John A McDonald; Todd D Camenisch
Journal:  Glycoconj J       Date:  2002 May-Jun       Impact factor: 2.916

8.  Prostate tumor cell exosomes containing hyaluronidase Hyal1 stimulate prostate stromal cell motility by engagement of FAK-mediated integrin signaling.

Authors:  Caitlin O McAtee; Christine Booth; Christian Elowsky; Lei Zhao; Jeremy Payne; Teresa Fangman; Steve Caplan; Michael D Henry; Melanie A Simpson
Journal:  Matrix Biol       Date:  2018-05-10       Impact factor: 11.583

9.  Spontaneous metastasis of prostate cancer is promoted by excess hyaluronan synthesis and processing.

Authors:  Alamelu G Bharadwaj; Joy L Kovar; Eileen Loughman; Christian Elowsky; Gregory G Oakley; Melanie A Simpson
Journal:  Am J Pathol       Date:  2009-02-13       Impact factor: 4.307

Review 10.  Emerging roles for hyaluronidase in cancer metastasis and therapy.

Authors:  Caitlin O McAtee; Joseph J Barycki; Melanie A Simpson
Journal:  Adv Cancer Res       Date:  2014       Impact factor: 6.242

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