Literature DB >> 12414783

In vitro detection of apoptosis in monocytes/macrophages infected with human coronavirus.

Arlene R Collins1.   

Abstract

Human coronavirus (HCoV) strain 229E infection, but not HCoV strain OC43 infection, of monocytes/macrophages from healthy donors and patients with multiple sclerosis in remission resulted in increased apoptosis, as measured by DNA changes and annexin V staining. Apoptosis correlated with the differential release of infectious virus. HCoV strain 229E titers were 10(3.5) to 10(6) 50% tissue culture-infective doses (TCID(50))/ml, and HCoV strain OC43 titers were only 10(1.2) to 10(2.7) TCID(50)/ml.

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Year:  2002        PMID： 12414783      PMCID： PMC130109          DOI： 10.1128/cdli.9.6.1392-1395.2002

Source DB:  PubMed          Journal:  Clin Diagn Lab Immunol        ISSN： 1071-412X


  18 in total

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Authors:  Paul G Smyth; Stephen A Berman
Journal:  Biotechniques       Date:  2002-03       Impact factor: 1.993

Review 3.  Immunopathogenesis of haemorrhagic enteritis virus (HEV) in turkeys.

Authors:  S Rautenschlein; J M Sharma
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4.  Neutralization of the chemokine CXCL10 reduces inflammatory cell invasion and demyelination and improves neurological function in a viral model of multiple sclerosis.

Authors:  M T Liu; H S Keirstead; T E Lane
Journal:  J Immunol       Date:  2001-10-01       Impact factor: 5.422

Review 5.  The role of pulmonary intravascular macrophages in porcine reproductive and respiratory syndrome virus infection.

Authors:  R Thanawongnuwech; P G Halbur; E L Thacker
Journal:  Anim Health Res Rev       Date:  2000-12       Impact factor: 2.615

6.  Activation of human monocytes induces differential resistance to apoptosis with rapid down regulation of caspase-8/FLICE.

Authors:  L P Perera; T A Waldmann
Journal:  Proc Natl Acad Sci U S A       Date:  1998-11-24       Impact factor: 11.205

7.  Increased expression of neutral endopeptidase (NEP) and aminopeptidase N (APN) on peripheral blood mononuclear cells in patients with multiple sclerosis.

Authors:  J Ziaber; Z Baj; J Paśnik; H Chmielewski; H Tchórzewski
Journal:  Immunol Lett       Date:  2000-02-01       Impact factor: 3.685

8.  Reduced macrophage infiltration and demyelination in mice lacking the chemokine receptor CCR5 following infection with a neurotropic coronavirus.

Authors:  W G Glass; M T Liu; W A Kuziel; T E Lane
Journal:  Virology       Date:  2001-09-15       Impact factor: 3.616

9.  Comparison of immunofluorescence with monoclonal antibodies and RT-PCR for the detection of human coronaviruses 229E and OC43 in cell culture.

Authors:  J Sizun; N Arbour; P J Talbot
Journal:  J Virol Methods       Date:  1998-06       Impact factor: 2.014

10.  Activation of glial cells by human coronavirus OC43 infection.

Authors:  J A Edwards; F Denis; P J Talbot
Journal:  J Neuroimmunol       Date:  2000-08-01       Impact factor: 3.478
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  43 in total

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Journal:  J Clin Pathol       Date:  2004-03       Impact factor: 3.411

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Journal:  AJNR Am J Neuroradiol       Date:  2020-04-30       Impact factor: 3.825

4.  HCoV-229E infects and activates monocytes.

Authors:  Marc Desforges; Tina Miletti; Mylène Gagnon; Pierre J Talbot
Journal:  Adv Exp Med Biol       Date:  2006       Impact factor: 2.622

5.  A human coronavirus responsible for the common cold massively kills dendritic cells but not monocytes.

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6.  Newly synthesized surfactants for surface mannosylation of respirable SLN assemblies to target macrophages in tuberculosis therapy.

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7.  Severe acute respiratory syndrome coronavirus triggers apoptosis via protein kinase R but is resistant to its antiviral activity.

Authors:  Verena Krähling; David A Stein; Martin Spiegel; Friedemann Weber; Elke Mühlberger
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8.  Validation of a medium-throughput method for evaluation of intracellular growth of Mycobacterium tuberculosis.

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9.  SARS coronavirus spike protein-induced innate immune response occurs via activation of the NF-kappaB pathway in human monocyte macrophages in vitro.

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Journal:  Virus Res       Date:  2009-01-29       Impact factor: 3.303

10.  Comparison of the effects of three kinds of IgYs, (normal, nanoliposomal and nanoparticle conjugated), which are produced against the small domains of DR5 protein on cancer cells.

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