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Literature DB >> 12370269

Antineutrophil cytoplasmic antibody and vasculitis: much more than a disease marker.

Vivette D'Agati¹.

Abstract

Entities: Disease

Mesh：

Substances：

Year: 2002 PMID： 12370269 PMCID： PMC151160 DOI： 10.1172/JCI16699

Source DB: PubMed Journal: J Clin Invest ISSN： 0021-9738 Impact factor: 14.808

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19 in total

1. Autoantibodies to myeloperoxidase aggravate mild anti-glomerular-basement-membrane-mediated glomerular injury in the rat.

Authors: P Heeringa; E Brouwer; P A Klok; M G Huitema; J van den Born; J J Weening; C G Kallenberg
Journal: Am J Pathol Date: 1996-11 Impact factor: 4.307

2. In situ production of TNF-alpha, IL-1 beta and IL-2R in ANCA-positive glomerulonephritis.

Authors: I L Noronha; C Krüger; K Andrassy; E Ritz; R Waldherr
Journal: Kidney Int Date: 1993-03 Impact factor: 10.612

3. Segmental necrotising glomerulonephritis with antineutrophil antibody: possible arbovirus aetiology?

Authors: D J Davies; J E Moran; J F Niall; G B Ryan
Journal: Br Med J (Clin Res Ed) Date: 1982 Aug 28-Sep 4

4. Antineutrophil cytoplasmic autoantibodies specific for myeloperoxidase cause glomerulonephritis and vasculitis in mice.

Authors: Hong Xiao; Peter Heeringa; Peiqi Hu; Zhi Liu; Minglang Zhao; Yasuaki Aratani; Nobuyo Maeda; Ronald J Falk; J Charles Jennette
Journal: J Clin Invest Date: 2002-10 Impact factor: 14.808

5. Anti-neutrophil cytoplasmic autoantibodies induce neutrophils to degranulate and produce oxygen radicals in vitro.

Authors: R J Falk; R S Terrell; L A Charles; J C Jennette
Journal: Proc Natl Acad Sci U S A Date: 1990-06 Impact factor: 11.205

6. Cytotoxic effects of antibodies to proteinase 3 (C-ANCA) on human endothelial cells.

Authors: W J Mayet; A Schwarting; K H Meyer zum Büschenfelde
Journal: Clin Exp Immunol Date: 1994-09 Impact factor: 4.330

7. Genetic selection for crescent formation yields mouse strain with rapidly progressive glomerulonephritis and small vessel vasculitis.

Authors: K Kinjoh; M Kyogoku; R A Good
Journal: Proc Natl Acad Sci U S A Date: 1993-04-15 Impact factor: 11.205

Antineutrophil cytoplasmic antibody and vasculitis: much more than a disease marker.

1. Autoantibodies to myeloperoxidase aggravate mild anti-glomerular-basement-membrane-mediated glomerular injury in the rat.

2. In situ production of TNF-alpha, IL-1 beta and IL-2R in ANCA-positive glomerulonephritis.

3. Segmental necrotising glomerulonephritis with antineutrophil antibody: possible arbovirus aetiology?

4. Antineutrophil cytoplasmic autoantibodies specific for myeloperoxidase cause glomerulonephritis and vasculitis in mice.

5. Anti-neutrophil cytoplasmic autoantibodies induce neutrophils to degranulate and produce oxygen radicals in vitro.

6. Cytotoxic effects of antibodies to proteinase 3 (C-ANCA) on human endothelial cells.

7. Genetic selection for crescent formation yields mouse strain with rapidly progressive glomerulonephritis and small vessel vasculitis.

8. Aggravation of rat nephrotoxic serum nephritis by anti-myeloperoxidase antibodies.

9. Activation of granulocytes by anti-neutrophil cytoplasmic antibodies (ANCA): a Fc gamma RII-dependent process.

10. Antimyeloperoxidase-associated proliferative glomerulonephritis: an animal model.

1. New markers: urine xanthine oxidase and myeloperoxidase in the early detection of urinary tract infection.