Literature DB >> 12224520

Macrophage interactions.

C Guidi-Rontani1, M Mock.   

Abstract

B. anthracis virulence is the sum of the contributions of factors involved in toxicity, growth and persistence in the host. Recent data has revealed that the interactions between B. anthracis and macrophage is central to the B. anthracis pathogenesis. This review presents and describes tactics by which B. anthracis not only overcomes and avoids macrophages but also perverts the host defense immune system and defense-related products to its advantage. The understanding of the complex network of such interactions is likely to allow new therapeutic and preventative strategies to be developed.

Entities:  

Mesh:

Substances:

Year:  2002        PMID: 12224520     DOI: 10.1007/978-3-662-05767-4_6

Source DB:  PubMed          Journal:  Curr Top Microbiol Immunol        ISSN: 0070-217X            Impact factor:   4.291


  12 in total

1.  Neutralization of B. anthracis toxins during ex vivo phagocytosis.

Authors:  Olga Tarasenko; Ashley Scott; April Jones; Lee Soderberg; Pierre Alusta
Journal:  Glycoconj J       Date:  2012-09-15       Impact factor: 2.916

2.  Cryo-EM analysis of the organization of BclA and BxpB in the Bacillus anthracis exosporium.

Authors:  Cynthia M Rodenburg; Sylvia A McPherson; Charles L Turnbough; Terje Dokland
Journal:  J Struct Biol       Date:  2014-03-06       Impact factor: 2.867

3.  Glycoconjugates prevent B. anthracis toxin-induced cell death through binding while activating macrophages.

Authors:  Olga Tarasenko; Ashley Scott; Lee Soderberg; Pierre Alusta
Journal:  Glycoconj J       Date:  2011-12-02       Impact factor: 2.916

4.  Protective antigen as a correlative marker for anthrax in animal models.

Authors:  David Kobiler; Shay Weiss; Haim Levy; Morly Fisher; Adva Mechaly; Avi Pass; Zeev Altboum
Journal:  Infect Immun       Date:  2006-10       Impact factor: 3.441

5.  Sensor domains encoded in Bacillus anthracis virulence plasmids prevent sporulation by hijacking a sporulation sensor histidine kinase.

Authors:  Andrea K White; James A Hoch; Marcin Grynberg; Adam Godzik; Marta Perego
Journal:  J Bacteriol       Date:  2006-09       Impact factor: 3.490

6.  Rap phosphatase of virulence plasmid pXO1 inhibits Bacillus anthracis sporulation.

Authors:  Cristina Bongiorni; Ricarda Stoessel; Dorinda Shoemaker; Marta Perego
Journal:  J Bacteriol       Date:  2006-01       Impact factor: 3.490

7.  Cost-effectiveness comparison of response strategies to a large-scale anthrax attack on the chicago metropolitan area: impact of timing and surge capacity.

Authors:  Demetrios N Kyriacou; Debra Dobrez; Jorge P Parada; Justin M Steinberg; Adam Kahn; Charles L Bennett; Brian P Schmitt
Journal:  Biosecur Bioterror       Date:  2012-07-30

8.  Polymeric glycoconjugates protect and activate macrophages to promote killing of Bacillus cereus spores during phagocytosis.

Authors:  Olga Tarasenko; Elizabeth Burton; Lee Soderberg; Pierre Alusta
Journal:  Glycoconj J       Date:  2008-01-17       Impact factor: 2.916

9.  Killing of Bacillus spores is mediated by nitric oxide and nitric oxide synthase during glycoconjugate-enhanced phagocytosis.

Authors:  Olga Tarasenko; Ashley Scott; Lee Soderberg; Usha Ponnappan; Pierre Alusta
Journal:  Glycoconj J       Date:  2009-06-23       Impact factor: 2.916

10.  Bacillus anthracis spore interactions with mammalian cells: relationship between germination state and the outcome of in vitro.

Authors:  Ian M Gut; Batcha Tamilselvam; Angela M Prouty; Bojana Stojkovic; Stephanie Czeschin; Wilfred A van der Donk; Steven R Blanke
Journal:  BMC Microbiol       Date:  2011-02-28       Impact factor: 3.605

View more

北京卡尤迪生物科技股份有限公司 © 2022-2023.