Literature DB >> 12218380

Pro12Ala substitution in the peroxisome proliferator-activated receptor-gamma is associated with increased leptin levels in women with type-2 diabetes mellitus.

I Simón1, J Vendrell, C Gutiérrez, J M Fernández-Real, I Vendrell, Ll Gallart, R Fontova, C Richart.   

Abstract

OBJECTIVE: To analyze the relationship between the peroxisome proliferator-activated receptor-gamma (PPARgamma2) Pro12Ala variant and type-2 diabetes mellitus and its correlation with some cytokine determinants of insulin resistance such as tumor necrosis factor (TNF)-alpha and leptin.
METHODS: The PPARgamma2 Pro12Ala genetic polymorphism was studied in 167 type-2 diabetic patients and 63 healthy controls. Serum leptin and plasma-soluble TNF-R2 were measured.
RESULTS: Women carriers of the Pro12Ala mutation exhibited higher leptin levels than women non-carriers (median 31.4 vs. 17.5 ng/ml; p < 0.005). sTNF-R2 levels did not show differences between the two genotypes. Analysis by the multiple linear regression model of leptin-body mass index controlled by the PPARgamma2 genotype showed that leptin levels were determined by the Pro12Ala mutation in type-2 diabetic women but not in men.
CONCLUSIONS: PPARgamma2 seems to be implicated in leptin homeostasis in type-2 diabetic women. Copyright 2002 S. Karger AG, Basel

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Year:  2002        PMID: 12218380     DOI: 10.1159/000064490

Source DB:  PubMed          Journal:  Horm Res        ISSN: 0301-0163


  10 in total

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  10 in total

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