Literature DB >> 12216704

Mucosal immunity preservation with bombesin or glutamine is not dependent on mucosal addressin cell adhesion molecule-1 expression.

Ben L Zarzaur1, Shigeo Ikeda, Cheryl D Johnson, Tho Le, Gordon Sacks, Kenneth A Kudsk.   

Abstract

BACKGROUND: Mucosal addressin cell adhesion molecule-1 (MAdCAM-1) is an adhesion molecule that directs naive T and B cells into Peyer's patches for sensitization and distribution to intestinal and extraintestinal sites. With no enteral stimulation, its expression drops rapidly in association with reduced Peyer's patch cell populations and increases rapidly with reinstitution of enteral feeding. Because both glutamine (GLN) and bombesin (BBS) preserve mucosal immunity, this study examined whether they preserve MAdCAM-1 expression.
METHODS: In 2 separate experiments, animals were randomized to IV cannulation with chow, total parenteral nutrition (TPN), and (experiment 1) 15 microg/kg BBS 3 times per day or (experiment 2) an isocaloric, isonitrogenous 2% GLN-supplemented solution. After 5 days of feeding, MAdCAM-1 expression in Peyer's patches, spleen, and intestine was measured using a dual radiolabeled monoclonal antibody technique.
RESULTS: MAdCAM-1 expression was not significantly improved from TPN levels either with BBS or GLN supplementation. Levels of MAdCAM-1 expression remained unchanged in non-Peyer's patch sites.
CONCLUSIONS: Although MAdCAM-1 is considered the gateway molecule for cell entry into mucosal immunity, this does not seem to be the mechanism for mucosal immunity preservation in nonenterally fed mice receiving bombesin or glutamine.

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Year:  2002        PMID: 12216704     DOI: 10.1177/0148607102026005265

Source DB:  PubMed          Journal:  JPEN J Parenter Enteral Nutr        ISSN: 0148-6071            Impact factor:   4.016


  10 in total

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2.  Jonathan E Rhoads lecture: Of mice and men... and a few hundred rats.

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3.  Intestinal epithelial cell-derived interleukin-7: A mechanism for the alteration of intraepithelial lymphocytes in a mouse model of total parenteral nutrition.

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4.  Glutamine prevents total parenteral nutrition-associated changes to intraepithelial lymphocyte phenotype and function: a potential mechanism for the preservation of epithelial barrier function.

Authors:  Keisuke Nose; Hua Yang; Xiaoyi Sun; Satoko Nose; Hiroyuki Koga; Yongjia Feng; Eiichi Miyasaka; Daniel H Teitelbaum
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Review 5.  Nutrition and gut immunity.

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6.  Enteral feeding preserves mucosal immunity despite in vivo MAdCAM-1 blockade of lymphocyte homing.

Authors:  Shigeo Ikeda; Kenneth A Kudsk; Kazuhiko Fukatsu; Cheryl D Johnson; Tho Le; Shannon Reese; Ben L Zarzaur
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7.  Colonic enteric nervous system analysis during parenteral nutrition.

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Review 8.  Food fight! Parenteral nutrition, enteral stimulation and gut-derived mucosal immunity.

Authors:  Joshua L Hermsen; Yoshifumi Sano; Kenneth A Kudsk
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9.  Dissociation of E-cadherin and beta-catenin in a mouse model of total parenteral nutrition: a mechanism for the loss of epithelial cell proliferation and villus atrophy.

Authors:  Yongjia Feng; Xiaoyi Sun; Hua Yang; Daniel H Teitelbaum
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10.  Glutamine Improves Innate Immunity and Prevents Bacterial Enteroinvasion During Parenteral Nutrition.

Authors:  Xinying Wang; Joseph F Pierre; Aaron F Heneghan; Rebecca A Busch; Kenneth A Kudsk
Journal:  JPEN J Parenter Enteral Nutr       Date:  2014-05-16       Impact factor: 4.016

  10 in total

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