Literature DB >> 12172985

c-KIT-expressing Ewing tumour cells are insensitive to imatinib mesylate (STI571).

Marc Hotfilder1, Claudia Lanvers, Heribert Jürgens, Joachim Boos, Josef Vormoor.   

Abstract

PURPOSE: In order to determine whether Ewing tumour patients may be potential candidates for imatinib mesylate therapy, we analysed the expression of the currently known imatinib mesylate-sensitive tyrosine kinases and tested sensitivity to imatinib mesylate in a panel of eight Ewing tumour cell lines in vitro.
METHODS: Expression of the different tyrosine kinases was assessed by flow cytometry and RT-PCR. Sensitivity to imatinib mesylate was analysed using a standard MTT proliferation assay.
RESULTS: Flow cytometric and RT-PCR analyses in a panel of eight Ewing tumour cell lines demonstrated expression of several imatinib mesylate-sensitive tyrosine kinases, including c-KIT, platelet-derived growth factor receptor, c-ABL and c-ARG. However, in the MTT proliferation assay, all eight Ewing tumour cell lines were found to be resistant to imatinib mesylate at concentrations ranging from 0.1 to 10 micro M.
CONCLUSIONS: Despite the expression of imatinib mesylate-sensitive tyrosine kinases, Ewing tumour cells proved resistant to imatinib mesylate in vitro. This observation has implications for the selection of patients for experimental therapy with imatinib mesylate.

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Year:  2002        PMID: 12172985     DOI: 10.1007/s00280-002-0477-8

Source DB:  PubMed          Journal:  Cancer Chemother Pharmacol        ISSN: 0344-5704            Impact factor:   3.333


  7 in total

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2.  Targeted therapies for bone sarcomas.

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3.  MTT assays cannot be utilized to study the effects of STI571/Gleevec on the viability of solid tumor cell lines.

Authors:  Jonathan T Sims; Rina Plattner
Journal:  Cancer Chemother Pharmacol       Date:  2009-04-26       Impact factor: 3.333

4.  ABT-869 inhibits the proliferation of Ewing Sarcoma cells and suppresses platelet-derived growth factor receptor beta and c-KIT signaling pathways.

Authors:  Alan K Ikeda; Dejah R Judelson; Noah Federman; Keith B Glaser; Elliot M Landaw; Christopher T Denny; Kathleen M Sakamoto
Journal:  Mol Cancer Ther       Date:  2010-03-02       Impact factor: 6.261

5.  PI3K/AKT is involved in mediating survival signals that rescue Ewing tumour cells from fibroblast growth factor 2-induced cell death.

Authors:  M Hotfilder; P Sondermann; A Senss; F van Valen; H Jürgens; J Vormoor
Journal:  Br J Cancer       Date:  2005-02-28       Impact factor: 7.640

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Authors:  Nancy R McAllister; Stephen L Lessnick
Journal:  Curr Treat Options Oncol       Date:  2005-11

7.  Bone sarcomas: from biology to targeted therapies.

Authors:  Nathalie Gaspar; Angela Di Giannatale; Birgit Geoerger; Françoise Redini; Nadège Corradini; Natacha Enz-Werle; Franck Tirode; Perrine Marec-Berard; Jean-Claude Gentet; Valérie Laurence; Sophie Piperno-Neumann; Odile Oberlin; Laurence Brugieres
Journal:  Sarcoma       Date:  2012-11-27
  7 in total

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