Literature DB >> 12169926

Adenosine A2A analogue improves neurologic outcome after spinal cord trauma in the rabbit.

David C Cassada1, Curtis G Tribble, Jeffrey S Young, James J Gangemi, A Reza Gohari, Paris D Butler, Jayson M Rieger, Irving L Kron, Joel Linden, John A Kern.   

Abstract

BACKGROUND: ATL-146e, an adenosine A2A agonist, reduces paralysis after spinal cord ischemia-reperfusion. We hypothesized that systemic ATL-146e could improve neurologic outcome after blunt spinal cord trauma.
METHODS: Twenty rabbits survived a thoracic spinal cord impact of 30 g-cm. One group received 0.06 microg/kg/min ATL-146e for the first 3 hours after impact (A2A group), whereas a second group received saline carrier (T/C group). Neurologic outcome was measured using the Tarlov scale (0-5). Histologic sections from the A2A and T/C groups were compared for neuronal viability.
RESULTS: There was significant improvement in Tarlov scores of A2A animals compared with T/C animals at 12 hours (p = 0.007), with a trend toward improvement at 36 (p = 0.08) and 48 (p = 0.09) hours after injury. There was decreased neuronal attrition in A2A animals (p = 0.06).
CONCLUSION: Systemic ATL-146e given after spinal cord trauma results in improved neurologic outcome. Adenosine A2A agonists may hold promise as a rapidly acting alternative to steroids in the early treatment of the spinal cord injured patient.

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Year:  2002        PMID: 12169926     DOI: 10.1097/00005373-200208000-00005

Source DB:  PubMed          Journal:  J Trauma        ISSN: 0022-5282


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