Literature DB >> 12093955

Maternal bone lead as an independent risk factor for fetal neurotoxicity: a prospective study.

Ahmed Gomaa1, Howard Hu, David Bellinger, Joel Schwartz, Shirng-Wern Tsaih, Teresa Gonzalez-Cossio, Lourdes Schnaas, Karen Peterson, Antonio Aro, Mauricio Hernandez-Avila.   

Abstract

OBJECTIVE: A number of prospective studies have examined lead levels in umbilical cord blood at birth as predictors of infant mental development. Although several have found significant inverse associations, others have not. Measurement of lead levels in maternal bone, now recognized as the source of much fetal exposure, has the potential to serve as a better or complementary predictor of lead's effect on the fetus. Our objective was to compare lead levels in umbilical cord blood and maternal bone as independent predictors of infant mental development using a prospective design.
METHODS: We recruited women who were giving birth at 3 maternity hospitals in Mexico City that serve a homogeneous middle-class community. Umbilical cord blood lead levels were measured by graphite furnace atomic absorption spectroscopy, and maternal lead levels in cortical (tibial) and trabecular (patellar) bone were measured within 4 weeks of giving birth using a 109-Cd K-x-ray fluorescence instrument. At 24 months of age, each infant was assessed using the Bayley Scales of Infant Development-II (Spanish Version).
RESULTS: A total of 197 mother-infant pairs completed this portion of the study and had data on all variables of interest. After adjustment for other well-known determinants of infant neurodevelopment, including maternal age, IQ, and education; paternal education; marital status; breastfeeding duration; infant gender; and infant illness, lead levels in umbilical cord blood and trabecular bone were significantly, independently, and inversely associated with the Mental Development Index (MDI) scores of the Bayley Scale. In relation to the lowest quartile of trabecular bone lead, the second, third, and fourth quartiles were associated with 5.4-, 7.2-, and 6.5-point decrements in adjusted MDI scores. A 2-fold increase in cord blood lead level (eg, from 5 to 10 micro g/dL) was associated with a 3.1-point decrement in MDI score, which is comparable to the magnitude of effect seen in previous studies.
CONCLUSION: Higher maternal trabecular bone lead levels constitute an independent risk factor for impaired mental development in infants at 24 months of age. This effect is probably attributable to mobilization of maternal bone lead stores, a phenomenon that may constitute a significant public health problem in view of the long residence time of lead in bone.

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Year:  2002        PMID: 12093955     DOI: 10.1542/peds.110.1.110

Source DB:  PubMed          Journal:  Pediatrics        ISSN: 0031-4005            Impact factor:   7.124


  39 in total

1.  Maternal MTHFR genotype and haplotype predict deficits in early cognitive development in a lead-exposed birth cohort in Mexico City.

Authors:  J Richard Pilsner; Howard Hu; Robert O Wright; Katarzyna Kordas; Adrienne S Ettinger; Brisa N Sánchez; David Cantonwine; Alicia L Lazarus; Alejandra Cantoral; Lourdes Schnaas; Martha Maria Téllez-Rojo; Mauricio Hernández-Avila
Journal:  Am J Clin Nutr       Date:  2010-05-26       Impact factor: 7.045

2.  High prenatal and postnatal lead exposure associated lead encephalopathy in an infant.

Authors:  Sandeep Kumar Kanwal; Virendra Kumar
Journal:  Indian J Pediatr       Date:  2011-06-10       Impact factor: 1.967

3.  Early postnatal blood manganese levels and children's neurodevelopment.

Authors:  Birgit Claus Henn; Adrienne S Ettinger; Joel Schwartz; Martha María Téllez-Rojo; Héctor Lamadrid-Figueroa; Mauricio Hernández-Avila; Lourdes Schnaas; Chitra Amarasiriwardena; David C Bellinger; Howard Hu; Robert O Wright
Journal:  Epidemiology       Date:  2010-07       Impact factor: 4.822

Review 4.  Lead neurotoxicity: effects on brain nitric oxide synthase.

Authors:  Concepción Nava-Ruiz; Marisela Méndez-Armenta; Camilo Ríos
Journal:  J Mol Histol       Date:  2012-04-17       Impact factor: 2.611

5.  A dopamine receptor (DRD2) but not dopamine transporter (DAT1) gene polymorphism is associated with neurocognitive development of Mexican preschool children with lead exposure.

Authors:  Katarzyna Kordas; Adrienne S Ettinger; David C Bellinger; Lourdes Schnaas; Martha María Téllez Rojo; Mauricio Hernández-Avila; Howard Hu; Robert O Wright
Journal:  J Pediatr       Date:  2011-05-17       Impact factor: 4.406

6.  Blood lead levels among pregnant women: historical versus contemporaneous exposures.

Authors:  Marie Lynn Miranda; Sharon E Edwards; Geeta K Swamy; Christopher J Paul; Brian Neelon
Journal:  Int J Environ Res Public Health       Date:  2010-04-01       Impact factor: 3.390

7.  Herbal supplement use and blood lead levels of United States adults.

Authors:  Catherine Buettner; Kenneth J Mukamal; Paula Gardiner; Roger B Davis; Russell S Phillips; Murray A Mittleman
Journal:  J Gen Intern Med       Date:  2009-07-03       Impact factor: 5.128

8.  Maternal self-esteem, exposure to lead, and child neurodevelopment.

Authors:  Pamela J Surkan; Lourdes Schnaas; Rosalind J Wright; Martha M Téllez-Rojo; Héctor Lamadrid-Figueroa; Howard Hu; Mauricio Hernández-Avila; David C Bellinger; Joel Schwartz; Estela Perroni; Robert O Wright
Journal:  Neurotoxicology       Date:  2007-12-15       Impact factor: 4.294

9.  Role of chelation during pregnancy in the lead poisoned patient.

Authors:  Mary Jean Brown
Journal:  J Med Toxicol       Date:  2013-12

10.  The U.S. Food and Drug Administration risk assessment on lead in women's and children's vitamins is based on outdated assumptions.

Authors:  Amir Miodovnik; Philip J Landrigan
Journal:  Environ Health Perspect       Date:  2009-03-25       Impact factor: 9.031

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