Literature DB >> 12084174

Novel 3'Rhesus box sequences confound RHD zygosity assignment.

Kimberly A Matheson1, Gregory A Denomme.   

Abstract

BACKGROUND: Paternal RHD zygosity is required for genetic counseling and management of HDN caused by anti-D. The most common D- haplotype is due to the deletion of RHD, which results in the formation of a hybrid Rhesus box, theoretically through the recombination of 5' and 3'Rhesus boxes. STUDY DESIGN AND METHODS: The validity of Rhesus box PstI analysis was assessed to determine RHD zygosity by correlating D phenotype, most probable genotype, and Rhesus box PCR-RFLP. RHD exons were examined, and a 501-bp Rhesus box fragment was sequenced that flanked the polymorphic PstI site.
RESULTS: Rhesus box analysis and the most probable genotype differed for 60 of 200 of the samples (30%). The incorrect zygosity assignment by the most probable genotype method is the likely reason for the difference. However, 8 of 328 samples showed Rhesus box copy numbers that were inconsistent with the D phenotype. Two D- samples with one hybrid Rhesus box had a nonfunctional RHD. Six D+ samples appeared to have two copies of the hybrid Rhesus box due to novel 3'Rhesus boxes that contained nucleotide polymorphisms previously assigned to the 5' and hybrid Rhesus boxes. All eight samples were from people of black descent, as determined by the GATA-1 silencing mutation at the FY locus.
CONCLUSION: Rhesus box PCR-RFLP analysis for RHD zygosity assignment is confounded by the presence of nonfunctional RHD+ (2.3% of D-) and novel, low frequency (0.9% of all alleles) 3'Rhesus box sequences.

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Year:  2002        PMID: 12084174     DOI: 10.1046/j.1537-2995.2002.00078.x

Source DB:  PubMed          Journal:  Transfusion        ISSN: 0041-1132            Impact factor:   3.157


  5 in total

1.  The low-prevalence Rh antigen STEM (RH49) is encoded by two different RHCE*ce818T alleles that are often in cis to RHD*DOL.

Authors:  Marion E Reid; Christine Halter Hipsky; Kim Hue-Roye; Gail Coghlan; Coral Olsen; Christine Lomas-Francis
Journal:  Transfusion       Date:  2012-06-28       Impact factor: 3.157

2.  RHCE*ceTI encodes partial c and partial e and is often in cis to RHD*DIVa.

Authors:  Connie M Westhoff; Sunitha Vege; Christine Halter Hipsky; Kim Hue-Roye; Tamara Copeland; Randall W Velliquette; Trina Horn; Christine Lomas-Francis; Marion E Reid
Journal:  Transfusion       Date:  2012-07-13       Impact factor: 3.157

3.  A novel laboratory technique demonstrating the influences of RHD zygosity and the RhCcEe phenotype on erythrocyte D antigen expression.

Authors:  Patrick T McGann; Jenny M Despotovic; Thad A Howard; Russell E Ware
Journal:  Am J Hematol       Date:  2011-11-25       Impact factor: 10.047

4.  Validated Reference Panel from Renewable Source of Genomic DNA Available for Standardization of Blood Group Genotyping.

Authors:  Evgeniya Volkova; Emilia Sippert; Meihong Liu; Teresita Mercado; Gregory A Denomme; Orieji Illoh; Zhugong Liu; Maria Rios
Journal:  J Mol Diagn       Date:  2019-03-12       Impact factor: 5.568

5.  RHD allele distribution in Africans of Mali.

Authors:  Franz F Wagner; Joann M Moulds; Anatole Tounkara; Bourema Kouriba; Willy A Flegel
Journal:  BMC Genet       Date:  2003-09-24       Impact factor: 2.797

  5 in total

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