| Literature DB >> 12077924 |
László Novák1, Miklós Emri, László Balkay, Sándor Szabó, László Rózsa, Péter Molnár.
Abstract
Ruptured aneurysms cause subarachnoidal hemorrhage (SAH) that may or may not result in neurological sequelae. Acute SAH has been shown to reduce cerebral blood flow and metabolism. An important question is whether or not the SAH induced vasospasm without clinical symptoms leads to cerebral ischemia? Using postoperative [18F]-fluorodeoxyglucose positron emission tomography in patients with vasospasm diagnosed by transcranial Doppler without clinical changes, hypermetabolism was detected as an indicator of diffuse compensated ischemia in 6 cases. In 2 patients with neurological deficit reduced glucose metabolism was observed in the frontal and temporal-basal cortex.Entities:
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Year: 2002 PMID: 12077924
Source DB: PubMed Journal: Orv Hetil ISSN: 0030-6002 Impact factor: 0.540