Literature DB >> 12077058

Visceral perception: inflammatory and non-inflammatory mediators.

L Bueno1, J Fioramonti.   

Abstract

Visceral hypersensitivity is currently the most widely accepted mechanism responsible for abdominal pain. Inflammatory mediators are known to sensitise primary afferents and to recruit silent nociceptors. Recent evidence suggests that non-inflammatory mediators also have the potential to trigger visceral pain. This sequence of events may constitute part of an alerting system which prompts the central nervous system to correct gastrointestinal responses to ingestion.

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Year:  2002        PMID： 12077058      PMCID： PMC1867723          DOI： 10.1136/gut.51.suppl_1.i19

Source DB:  PubMed          Journal:  Gut        ISSN： 0017-5749            Impact factor:   23.059


  28 in total

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Review 4.  Pathobiology of visceral pain: molecular mechanisms and therapeutic implications. III. Visceral afferent pathways: a source of new therapeutic targets for abdominal pain.

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Journal:  Br J Pharmacol       Date:  1990-07       Impact factor: 8.739

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  34 in total

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4.  The effect of spinal cord injury on the neurochemical properties of vagal sensory neurons.

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5.  The effect of curcumin on the brain-gut axis in rat model of irritable bowel syndrome: involvement of 5-HT-dependent signaling.

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6.  Current insights in to the pathophysiology of Irritable Bowel Syndrome.

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Review 7.  Is irritable bowel syndrome an organic disorder?

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8.  Increased colonic pain sensitivity in irritable bowel syndrome is the result of an increased tendency to report pain rather than increased neurosensory sensitivity.

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Review 10.  Modulation of visceral pain and inflammation by protease-activated receptors.

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