Literature DB >> 12072906

Tissue renin-angiotensin system and end-organ damage.

Friedrich C Luft1.   

Abstract

Entities:  

Mesh：

Substances：

Year:  2002        PMID： 12072906     DOI： 10.1007/s00109-002-0334-6

Source DB:  PubMed          Journal:  J Mol Med (Berl)        ISSN： 0946-2716            Impact factor:   4.599


× No keyword cloud information.
  7 in total

1.  History about the discovery of the renin-angiotensin system.

Authors:  N Basso; N A Terragno
Journal:  Hypertension       Date:  2001-12-01       Impact factor: 10.190

2.  Urinary excretion of angiotensinogen reflects intrarenal angiotensinogen production.

Authors:  Hiroyuki Kobori; Lisa M Harrison-Bernard; L Gabriel Navar
Journal:  Kidney Int       Date:  2002-02       Impact factor: 10.612

3.  Angiotensinogen and its cleaved derivatives inhibit angiogenesis.

Authors:  Jérôme Célérier; Amauri Cruz; Noël Lamandé; Jean-Marie Gasc; Pierre Corvol
Journal:  Hypertension       Date:  2002-02       Impact factor: 10.190

4.  Evidence of renin release or production in splanchnic territory.

Authors:  D Ganten; K Hayduk; H M Brecht; R Boucher; J Genest
Journal:  Nature       Date:  1970-05-09       Impact factor: 49.962

5.  Reduced hypertension-induced end-organ damage in mice lacking cardiac and renal angiotensinogen synthesis.

Authors:  Ningling Kang; Thomas Walther; Xiao-Li Tian; Jürgen Bohlender; Akiyoshi Fukamizu; Detlev Ganten; Michael Bader
Journal:  J Mol Med (Berl)       Date:  2002-03-14       Impact factor: 4.599

6.  Angiotensin-forming enzyme in brain tissue.

Authors:  D Ganten; J L Minnich; P Granger; K Hayduk; H M Brecht; A Barbeau; R Boucher; J Genest
Journal:  Science       Date:  1971-07-02       Impact factor: 47.728

Review 7.  Renin is not synthesized by cardiac and extrarenal vascular tissues. A review of experimental evidence.

Authors:  N von Lutterotti; D F Catanzaro; J E Sealey; J H Laragh
Journal:  Circulation       Date:  1994-01       Impact factor: 29.690
  7 in total
  1 in total

Review 1.  ACE-2 down-regulation may act as a transient molecular disease causing RAAS dysregulation and tissue damage in the microcirculatory environment among COVID-19 patients.

Authors:  Simone Gusmão Ramos; Bruna Amanda da Cruz Rattis; Giulia Ottaviani; Mara Rubia Nunes Celes; Eliane Pedra Dias
Journal:  Am J Pathol       Date:  2021-05-05       Impact factor: 4.307
  1 in total

北京卡尤迪生物科技股份有限公司 © 2022-2023.